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本文引用的文献
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Molecular classification of sporadic Creutzfeldt-Jakob disease.
Brain. 2003 Jun;126(Pt 6):1333-46. doi: 10.1093/brain/awg125.
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BSE prions propagate as either variant CJD-like or sporadic CJD-like prion strains in transgenic mice expressing human prion protein.
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Differentiation of prion protein glycoforms from naturally occurring sheep scrapie, sheep-passaged scrapie strains (CH1641 and SSBP1), bovine spongiform encephalopathy (BSE) cases and Romney and Cheviot breed sheep experimentally inoculated with BSE using two monoclonal antibodies.
Acta Neuropathol. 2002 Sep;104(3):279-86. doi: 10.1007/s00401-002-0556-2. Epub 2002 Jun 26.
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Targeted surveillance to assess the prevalence of BSE in high-risk populations in western France and the associated risk factors.
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Identification of inter-species transmission of prion strains.
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Molecular analysis of the abnormal prion protein during coinfection of mice by bovine spongiform encephalopathy and a scrapie agent.
J Virol. 2001 Jan;75(1):107-14. doi: 10.1128/JVI.75.1.107-114.2001.
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Comparison of French natural scrapie isolates with bovine spongiform encephalopathy and experimental scrapie infected sheep.
Neurosci Lett. 2000 Apr 28;284(3):175-8. doi: 10.1016/s0304-3940(00)01047-8.
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Efficiency of Western blotting for the specific immunodetection of proteinase K-resistant prion protein in BSE diagnosis in France.
Vet Rec. 2000 Jan 15;146(3):74-6. doi: 10.1136/vr.146.3.74.
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Compelling transgenetic evidence for transmission of bovine spongiform encephalopathy prions to humans.
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Strain-specific prion-protein conformation determined by metal ions.
Nat Cell Biol. 1999 May;1(1):55-9. doi: 10.1038/9030.
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