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本文引用的文献
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Ovarian insufficiency and early pregnancy loss induced by activation of the innate immune system.
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Cytokines and steroidogenesis.
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Regulatory T cells mediate maternal tolerance to the fetus.
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Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome.
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Regulation by C5a of neutrophil activation during sepsis.
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At the innate frontiers between mother and fetus: linking abortion with complement activation.
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Complement and dilated cardiomyopathy: a role of sublytic terminal complement complex-induced tumor necrosis factor-alpha synthesis in cardiac myocytes.
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Complement C3 activation is required for antiphospholipid antibody-induced fetal loss.
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Cytokine promoter gene polymorphisms and idiopathic recurrent pregnancy loss.
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