Soria E D, Fine E J
Department of Neurology, School of Medicine, State University of New York, Buffalo.
Electromyogr Clin Neurophysiol. 1992 Jan-Feb;32(1-2):63-71.
10 patients with chronic sequelae from vitamin B12 deficiency showed a characteristic profile of abnormalities of median and peroneal somatosensory evoked potentials: mild to moderately slow peripheral conduction to Erb's point, normal conduction or mild slowing across the cervical portion of the median somatosensory evoked potentials, and absence or severe slowing of impulse propagation along the spinal cord with the peroneal somatosensory evoked potentials. These findings are compared with those of other published series of somatosensory evoked potentials in vitamin B12 deficiency. These results shed light on the chronic physiological and anatomical residuals in the nervous system of patients treated for vitamin B12 deficiency.
10例维生素B12缺乏慢性后遗症患者表现出正中神经和腓总神经体感诱发电位异常的特征性表现：外周至Erb点的传导轻度至中度减慢，正中神经体感诱发电位在颈部的传导正常或轻度减慢，而腓总神经体感诱发电位沿脊髓的冲动传导缺失或严重减慢。将这些发现与其他已发表的维生素B12缺乏患者体感诱发电位系列研究结果进行比较。这些结果揭示了维生素B12缺乏症治疗患者神经系统中的慢性生理和解剖学残留情况。
