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血小板5-羟色胺能标志物作为强迫症的内表型

Platelet serotonergic markers as endophenotypes for obsessive-compulsive disorder.

作者信息

Delorme Richard, Betancur Catalina, Callebert Jacques, Chabane Nadia, Laplanche Jean-Louis, Mouren-Simeoni Marie-Christine, Launay Jean-Marie, Leboyer Marion

机构信息

INSERM U 513, Faculté de Médecine, Créteil, France.

出版信息

Neuropsychopharmacology. 2005 Aug;30(8):1539-47. doi: 10.1038/sj.npp.1300752.

Abstract

Although compelling evidence has shown that obsessive-compulsive disorder (OCD) has a strong genetic component, its genetic basis remains to be elucidated. Identifying biological abnormalities in nonaffected relatives is one of the strategies advocated to isolate genetic vulnerability factors in complex disorders. Since peripheral serotonergic disturbances are frequently observed in OCD patients, the aim of this study was to investigate if they could represent endophenotypes, by searching for similar abnormalities in the unaffected parents of OCD patients. We assessed whole blood serotonin (5-HT) concentration, platelet 5-HT transporter (5-HTT) and 5-HT2A receptor-binding characteristics, and platelet inositol trisphosphate (IP3) content in a sample of OCD probands (n = 48) and their unaffected parents (n = 65), and compared them with sex- and age-matched controls (n = 113). Lower whole blood 5-HT concentration, fewer platelet 5-HTT-binding sites, and higher platelet IP3 content were found in OCD probands and their unaffected parents compared to controls. Whole blood 5-HT concentration showed a strong correlation within families (p < 0.001). The only parameter that appeared to discriminate affected and unaffected subjects was 5-HT2A receptor-binding characteristics, with increased receptor number and affinity in parents and no change in OCD probands. The presence of peripheral serotonergic abnormalities in OCD patients and their unaffected parents supports a familial origin of these disturbances. These alterations may serve as endophenotypic markers in OCD, and could contribute to the study of the biological mechanisms and genetic underpinnings of the disorder.

摘要

尽管有力的证据表明强迫症(OCD)有很强的遗传成分,但其遗传基础仍有待阐明。识别未患病亲属中的生物学异常是在复杂疾病中分离遗传易感性因素所倡导的策略之一。由于在强迫症患者中经常观察到外周血清素紊乱,本研究的目的是通过在强迫症患者未患病的父母中寻找类似异常,来调查这些异常是否可代表内表型。我们评估了48例强迫症先证者及其65例未患病父母的全血血清素(5-HT)浓度、血小板5-HT转运体(5-HTT)和5-HT2A受体结合特性以及血小板三磷酸肌醇(IP3)含量,并将他们与性别和年龄匹配的113名对照进行比较。与对照组相比,强迫症先证者及其未患病父母的全血5-HT浓度较低、血小板5-HTT结合位点较少且血小板IP3含量较高。全血5-HT浓度在家族内部显示出很强的相关性(p < 0.001)。唯一似乎能区分患病和未患病个体的参数是5-HT2A受体结合特性,父母的受体数量和亲和力增加,而强迫症先证者无变化。强迫症患者及其未患病父母存在外周血清素异常,这支持了这些紊乱的家族起源。这些改变可能作为强迫症的内表型标记,并有助于该疾病生物学机制和遗传基础的研究。

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