Thind P, Brandt B, Kristensen J K
Department of Urology, Rigshospitalet, University of Copenhagen, Denmark.
Urol Int. 1992;48(3):320-2.
Fifteen men with a recent attack of acute epididymitis were examined urodynamically and endoscopically after the symptoms had resolved. Thirteen of the 15 patients had abnormal voiding. Two patients had stricture formation in the posterior urethra, one had benign prostatic hyperplasia, one had bladder neck sclerosis, and 4 had detrusor-external sphincter dyssynergia. Three patients including the one with prostatic enlargement presented abdominal straining exceeding 50 cm H2O during micturition. The cause of an obstructive voiding pattern could not be established in 3 patients. The results support the theory of urethrovasal reflux due to a high hydrostatic pressure at the collicular level as a pathophysiological factor in acute epididymitis.
15名近期患有急性附睾炎的男性在症状消退后接受了尿动力学和内镜检查。15名患者中有13名排尿异常。2名患者后尿道出现狭窄,1名患有良性前列腺增生,1名患有膀胱颈硬化,4名患有逼尿肌-外括约肌协同失调。包括前列腺肿大患者在内的3名患者排尿时腹部压力超过50 cm H2O。3名患者未明确梗阻性排尿模式的原因。这些结果支持了如下理论:作为急性附睾炎的病理生理因素,在精阜水平的高静水压导致尿道输精管反流。