大肠杆菌的Mg2 + -ATP酶缺陷型突变体与硫胺素转运
Mg2+-ATPase defective mutant of Escherichia coli and thiamine transport.
作者信息
Nishimune T, Hayashi R
出版信息
Experientia. 1979 Oct 15;35(10):1318-20. doi: 10.1007/BF01963982.
PMID:159188
Abstract
Mg2+-ATPase deficient mutant of Escherichia coli showed an evident dependency of thiamine uptake on the oxidative metabolism of glucose, whereas the parent strain did not. In both cells, this uptake was completely inhibited by H+ conductors.
摘要
大肠杆菌的Mg2 + -ATP酶缺陷型突变体显示硫胺素摄取对葡萄糖氧化代谢有明显依赖性，而亲本菌株则没有。在这两种细胞中，这种摄取都被H +导体完全抑制。
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