相似文献
1
Nogo-B is a new physiological substrate for MAPKAP-K2.
Biochem J. 2005 Oct 15;391(Pt 2):433-40. doi: 10.1042/BJ20050935.
2
Inhibition of SAPK2a/p38 prevents hnRNP A0 phosphorylation by MAPKAP-K2 and its interaction with cytokine mRNAs.
EMBO J. 2002 Dec 2;21(23):6505-14. doi: 10.1093/emboj/cdf639.
3
The phosphorylation of CapZ-interacting protein (CapZIP) by stress-activated protein kinases triggers its dissociation from CapZ.
Biochem J. 2005 Jul 1;389(Pt 1):127-35. doi: 10.1042/BJ20050387.
4
Identification of glycogen synthase as a new substrate for stress-activated protein kinase 2b/p38beta.
Biochem J. 2004 Apr 1;379(Pt 1):133-9. doi: 10.1042/BJ20031559.
5
Mitogen- and stress-activated protein kinase-1 (MSK1) is directly activated by MAPK and SAPK2/p38, and may mediate activation of CREB.
EMBO J. 1998 Aug 3;17(15):4426-41. doi: 10.1093/emboj/17.15.4426.
6
Identification of anisomycin-activated kinases p45 and p55 in murine cells as MAPKAP kinase-2.
Oncogene. 1996 Feb 15;12(4):805-12.
7
CXCL12 and C5a trigger cell migration via a PAK1/2-p38alpha MAPK-MAPKAP-K2-HSP27 pathway.
Cell Signal. 2006 Nov;18(11):1897-905. doi: 10.1016/j.cellsig.2006.02.006. Epub 2006 Mar 6.
8
Activation of the novel stress-activated protein kinase SAPK4 by cytokines and cellular stresses is mediated by SKK3 (MKK6); comparison of its substrate specificity with that of other SAP kinases.
EMBO J. 1997 Jun 16;16(12):3563-71. doi: 10.1093/emboj/16.12.3563.
9
Stress stimuli increase calcium-induced arachidonic acid release through phosphorylation of cytosolic phospholipase A2.
Biochem J. 1999 Dec 1;344 Pt 2(Pt 2):359-66.
10
Endoplasmic reticulum-associated ubiquitin-conjugating enzyme Ube2j1 is a novel substrate of MK2 (MAPKAP kinase-2) involved in MK2-mediated TNFα production.
Biochem J. 2013 Dec 1;456(2):163-72. doi: 10.1042/BJ20130755.
引用本文的文献
1
TgMORN2, a MORN Family Protein Involved in the Regulation of Endoplasmic Reticulum Stress in .
Int J Mol Sci. 2023 Jun 16;24(12):10228. doi: 10.3390/ijms241210228.
2
Nogo-A reduces ceramide de novo biosynthesis to protect from heart failure.
Cardiovasc Res. 2023 Mar 31;119(2):506-519. doi: 10.1093/cvr/cvac108.
3
Mitogen-activated protein kinase-activated protein kinase-2 (MK2) and its role in cell survival, inflammatory signaling, and migration in promoting cancer.
Mol Carcinog. 2022 Feb;61(2):173-199. doi: 10.1002/mc.23348. Epub 2021 Sep 24.
4
A Novel Role of Nogo Proteins: Regulating Macrophages in Inflammatory Disease.
Cell Mol Neurobiol. 2022 Nov;42(8):2439-2448. doi: 10.1007/s10571-021-01124-0. Epub 2021 Jul 5.
5
Osmotic Stress Blocks Mobility and Dynamic Regulation of Centriolar Satellites.
Cells. 2018 Jun 22;7(7):65. doi: 10.3390/cells7070065.
6
Platelet procoagulant phenotype is modulated by a p38-MK2 axis that regulates RTN4/Nogo proximal to the endoplasmic reticulum: utility of pathway analysis.
Am J Physiol Cell Physiol. 2018 May 1;314(5):C603-C615. doi: 10.1152/ajpcell.00177.2017. Epub 2018 Feb 7.
7
Ras transformation results in cleavage of reticulon protein Nogo-B that is associated with impairment of IFN response.
Cell Cycle. 2015;14(14):2301-10. doi: 10.1080/15384101.2015.1044187. Epub 2015 May 6.
8
p38MAPK/MK2-mediated phosphorylation of RBM7 regulates the human nuclear exosome targeting complex.
RNA. 2015 Feb;21(2):262-78. doi: 10.1261/rna.048090.114. Epub 2014 Dec 18.
9
Nogo-C contributes to HCC tumorigenesis via suppressing cell growth and its interactome analysis with comparative proteomics research.
Int J Clin Exp Pathol. 2014 Apr 15;7(5):2044-55. eCollection 2014.
10
The p38/MK2-driven exchange between tristetraprolin and HuR regulates AU-rich element-dependent translation.
PLoS Genet. 2012 Sep;8(9):e1002977. doi: 10.1371/journal.pgen.1002977. Epub 2012 Sep 27.
本文引用的文献
1
The search for physiological substrates of MAP and SAP kinases in mammalian cells.
Trends Cell Biol. 1997 Sep;7(9):353-61. doi: 10.1016/S0962-8924(97)01105-7.
2
The phosphorylation of CapZ-interacting protein (CapZIP) by stress-activated protein kinases triggers its dissociation from CapZ.
Biochem J. 2005 Jul 1;389(Pt 1):127-35. doi: 10.1042/BJ20050387.
3
Genetic deletion of the Nogo receptor does not reduce neurite inhibition in vitro or promote corticospinal tract regeneration in vivo.
Proc Natl Acad Sci U S A. 2005 Jan 25;102(4):1205-10. doi: 10.1073/pnas.0409026102. Epub 2005 Jan 12.
4
Nogo-A, -B, and -C are found on the cell surface and interact together in many different cell types.
J Biol Chem. 2005 Apr 1;280(13):12494-502. doi: 10.1074/jbc.M411827200. Epub 2005 Jan 7.
5
A new role for Nogo as a regulator of vascular remodeling.
Nat Med. 2004 Apr;10(4):382-8. doi: 10.1038/nm1020. Epub 2004 Mar 21.
6
Feedback control of the protein kinase TAK1 by SAPK2a/p38alpha.
EMBO J. 2003 Nov 3;22(21):5793-805. doi: 10.1093/emboj/cdg552.
7
HIF prolyl-hydroxylase 2 is the key oxygen sensor setting low steady-state levels of HIF-1alpha in normoxia.
EMBO J. 2003 Aug 15;22(16):4082-90. doi: 10.1093/emboj/cdg392.
8
Lack of enhanced spinal regeneration in Nogo-deficient mice.
Neuron. 2003 Apr 24;38(2):213-24. doi: 10.1016/s0896-6273(03)00225-3.
9
Systemic deletion of the myelin-associated outgrowth inhibitor Nogo-A improves regenerative and plastic responses after spinal cord injury.
Neuron. 2003 Apr 24;38(2):201-11. doi: 10.1016/s0896-6273(03)00226-5.
10
Axon regeneration in young adult mice lacking Nogo-A/B.
Neuron. 2003 Apr 24;38(2):187-99. doi: 10.1016/s0896-6273(03)00147-8.
Zotero 插件