Huttner Hagen B, Berger Christian, Schwab Stefan
Department of Neurology, University of Heidelberg, Heidelberg, Germany.
Neurocrit Care. 2005;3(2):171-3. doi: 10.1385/NCC:3:2:171.
We report a patient with severe ethylene glycol poisoning initially mimicking acute basilar artery occlusion and elucidate the importance of immediate diagnosis and treatment: a previously healthy 59-year-old truck driver presenting with hallmarks of basilar artery syndrome after having consumed an unknown substance. Immediate application of intravenous ethanol and hemodialysis could not prevent the development of a malignant brain edema within hours. This report describes a new clinical presentation of severe ethylene glycol intoxication mimicking acute basilar artery occlusion with the development of a fatal brain edema within hours, despite adequate treatment.
我们报告了一名严重乙二醇中毒患者，其最初表现类似急性基底动脉闭塞，并阐明了立即诊断和治疗的重要性：一名此前健康的59岁卡车司机，在摄入不明物质后出现基底动脉综合征的特征。立即静脉应用乙醇和进行血液透析未能阻止数小时内恶性脑水肿的发展。本报告描述了严重乙二醇中毒的一种新的临床表现，即类似急性基底动脉闭塞，尽管进行了充分治疗，但仍在数小时内发展为致命性脑水肿。
