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1
Strong BCL10 nuclear expression identifies gastric MALT lymphomas that do not respond to H pylori eradication.BCL10强核表达可识别对幽门螺杆菌根除治疗无反应的胃黏膜相关淋巴组织淋巴瘤。
Gut. 2006 Jan;55(1):137-8. doi: 10.1136/gut.2005.081117.
2
Nuclear expression of BCL10 or nuclear factor kappa B predicts Helicobacter pylori-independent status of early-stage, high-grade gastric mucosa-associated lymphoid tissue lymphomas.BCL10的核表达或核因子κB可预测早期、高级别胃黏膜相关淋巴组织淋巴瘤的非幽门螺杆菌依赖状态。
J Clin Oncol. 2004 Sep 1;22(17):3491-7. doi: 10.1200/JCO.2004.10.087.
3
Differential response to H. pylori eradication therapy of co-existing diffuse large B-cell lymphoma and MALT lymphoma of stomach-significance of tumour cell clonality and BCL10 expression.胃弥漫性大B细胞淋巴瘤与黏膜相关淋巴组织淋巴瘤共存时对幽门螺杆菌根除治疗的差异反应——肿瘤细胞克隆性和BCL10表达的意义
J Pathol. 2007 Feb;211(3):296-304. doi: 10.1002/path.2117.
4
BCL10 nuclear expression and t(11;18)(q21;q21) indicate nonresponsiveness to Helicobacter pylori eradication of Chinese primary gastric MALT lymphoma.BCL10核表达及t(11;18)(q21;q21)提示中国原发性胃黏膜相关淋巴组织淋巴瘤对幽门螺杆菌根除治疗无反应。
Int J Hematol. 2008 Dec;88(5):516-523. doi: 10.1007/s12185-008-0187-z. Epub 2008 Oct 24.
5
MALT lymphoma with t(14;18)(q32;q21)/IGH-MALT1 is characterized by strong cytoplasmic MALT1 and BCL10 expression.伴有t(14;18)(q32;q21)/IGH-MALT1的黏膜相关淋巴组织淋巴瘤的特征是细胞质MALT1和BCL10呈强表达。
J Pathol. 2005 Feb;205(3):293-301. doi: 10.1002/path.1715.
6
Etiology and therapy of Helicobacter pylori-associated gastric lymphomas.幽门螺杆菌相关胃淋巴瘤的病因与治疗
Ann Hematol. 2003 Sep;82(9):535-45. doi: 10.1007/s00277-003-0677-2. Epub 2003 Jul 3.
7
Gastric lymphomas: genetics and resistance to H. pylori eradication.胃淋巴瘤：遗传学与对幽门螺杆菌根除治疗的耐药性
Verh Dtsch Ges Pathol. 2003;87:116-22.
8
Nuclear expression of BCL10 or nuclear factor kappa B helps predict Helicobacter pylori-independent status of low-grade gastric mucosa-associated lymphoid tissue lymphomas with or without t(11;18)(q21;q21).BCL10的核表达或核因子κB有助于预测伴有或不伴有t(11;18)(q21;q21)的低级别胃黏膜相关淋巴组织淋巴瘤的幽门螺杆菌非依赖状态。
Blood. 2005 Aug 1;106(3):1037-41. doi: 10.1182/blood-2005-01-0004. Epub 2005 Apr 21.
9
[Long-term outcome in patients with gastric MALT lymphoma after H. pylori eradication].[幽门螺杆菌根除治疗后胃黏膜相关淋巴组织淋巴瘤患者的长期预后]
Nihon Rinsho. 2005 Nov;63 Suppl 11:516-9.
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[H. pylori-negative gastric MALT lymphoma].幽门螺杆菌阴性的胃黏膜相关淋巴组织淋巴瘤
Nihon Rinsho. 2005 Nov;63 Suppl 11:625-8.

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Role of non- gastric Helicobacters in -negative gastric mucosa-associated lymphoid tissue lymphoma.非胃型幽门螺杆菌在胃黏膜相关淋巴组织淋巴瘤中的作用。
World J Gastroenterol. 2023 Aug 28;29(32):4851-4859. doi: 10.3748/wjg.v29.i32.4851.
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Clinical relevance of molecular aspects in extranodal marginal zone lymphoma: a critical appraisal.结外边缘区淋巴瘤分子特征的临床相关性：批判性评估
Ther Adv Med Oncol. 2023 Jun 22;15:17588359231183565. doi: 10.1177/17588359231183565. eCollection 2023.
3
Recent Advances in the Genetic of MALT Lymphomas.黏膜相关淋巴组织淋巴瘤遗传学的最新进展
Cancers (Basel). 2021 Dec 30;14(1):176. doi: 10.3390/cancers14010176.
4
Contribution of nuclear BCL10 expression to tumor progression and poor prognosis of advanced and/or metastatic pancreatic ductal adenocarcinoma by activating NF-κB-related signaling.核内BCL10表达通过激活NF-κB相关信号通路促进晚期和/或转移性胰腺导管腺癌的肿瘤进展及预后不良。
Cancer Cell Int. 2021 Aug 19;21(1):436. doi: 10.1186/s12935-021-02143-z.
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First-line antibiotic therapy in Helicobacter pylori-negative low-grade gastric mucosa-associated lymphoid tissue lymphoma.幽门螺杆菌阴性低级别胃黏膜相关淋巴组织淋巴瘤的一线抗生素治疗。
Sci Rep. 2017 Oct 30;7(1):14333. doi: 10.1038/s41598-017-14102-8.
6
B-cell receptor signaling as a driver of lymphoma development and evolution.B 细胞受体信号作为淋巴瘤发生和演进的驱动因素。
Semin Cancer Biol. 2013 Dec;23(6):410-21. doi: 10.1016/j.semcancer.2013.09.001. Epub 2013 Sep 20.
7
Detection of the Helicobacter pylori CagA protein in gastric mucosa-associated lymphoid tissue lymphoma cells: clinical and biological significance.检测胃黏膜相关淋巴组织淋巴瘤细胞中的幽门螺杆菌 CagA 蛋白：临床和生物学意义。
Blood Cancer J. 2013 Jul 12;3(7):e125. doi: 10.1038/bcj.2013.22.
8
Lipopolysaccharide-induced activation of NF-κB non-canonical pathway requires BCL10 serine 138 and NIK phosphorylations.脂多糖诱导的 NF-κB 非经典途径的激活需要 BCL10 丝氨酸 138 和 NIK 的磷酸化。
Exp Cell Res. 2010 Nov 15;316(19):3317-27. doi: 10.1016/j.yexcr.2010.05.004. Epub 2010 May 11.
9
Emu-BCL10 mice exhibit constitutive activation of both canonical and noncanonical NF-kappaB pathways generating marginal zone (MZ) B-cell expansion as a precursor to splenic MZ lymphoma.鸸鹋-BCL10小鼠表现出经典和非经典NF-κB信号通路的组成性激活，导致边缘区（MZ）B细胞扩增，这是脾MZ淋巴瘤的前兆。
Blood. 2009 Nov 5;114(19):4158-68. doi: 10.1182/blood-2008-12-192583. Epub 2009 Aug 20.
10
Pulmonary mucosa-associated lymphoid tissue lymphoma with strong nuclear B-cell CLL/lymphoma 10 (BCL10) expression and novel translocation t(1;2)(p22;p12)/immunoglobulin kappa chain-BCL10.具有强核B细胞慢性淋巴细胞白血病/淋巴瘤10（BCL10）表达及新型易位t(1;2)(p22;p12)/免疫球蛋白κ链-BCL10的肺黏膜相关淋巴组织淋巴瘤
J Clin Pathol. 2007 Jun;60(6):727-8. doi: 10.1136/jcp.2006.041012. Epub 2006 Nov 10.

本文引用的文献

1
MALT lymphoma with t(14;18)(q32;q21)/IGH-MALT1 is characterized by strong cytoplasmic MALT1 and BCL10 expression.伴有t(14;18)(q32;q21)/IGH-MALT1的黏膜相关淋巴组织淋巴瘤的特征是细胞质MALT1和BCL10呈强表达。
J Pathol. 2005 Feb;205(3):293-301. doi: 10.1002/path.1715.
2
MALT lymphoma: from morphology to molecules.黏膜相关淋巴组织淋巴瘤：从形态学到分子层面
Nat Rev Cancer. 2004 Aug;4(8):644-53. doi: 10.1038/nrc1409.
3
Variable frequencies of t(11;18)(q21;q21) in MALT lymphomas of different sites: significant association with CagA strains of H pylori in gastric MALT lymphoma.不同部位MALT淋巴瘤中t(11;18)(q21;q21)的可变频率：与胃MALT淋巴瘤中幽门螺杆菌CagA菌株显著相关。
Blood. 2003 Aug 1;102(3):1012-8. doi: 10.1182/blood-2002-11-3502. Epub 2003 Apr 3.
4
T(11;18) is a marker for all stage gastric MALT lymphomas that will not respond to H. pylori eradication.T(11;18)是所有阶段胃黏膜相关淋巴组织淋巴瘤的一个标志物，这类淋巴瘤对根除幽门螺杆菌无反应。
Gastroenterology. 2002 May;122(5):1286-94. doi: 10.1053/gast.2002.33047.
5
Predictive value of endoscopic ultrasonography for regression of gastric low grade and high grade MALT lymphomas after eradication of Helicobacter pylori.根除幽门螺杆菌后内镜超声检查对胃低级别和高级别黏膜相关淋巴组织淋巴瘤消退的预测价值
Gut. 2001 Apr;48(4):454-60. doi: 10.1136/gut.48.4.454.
6
Predictive factors for regression of gastric MALT lymphoma after anti-Helicobacter pylori treatment.抗幽门螺杆菌治疗后胃黏膜相关淋巴组织淋巴瘤消退的预测因素。
Gut. 2001 Mar;48(3):297-303. doi: 10.1136/gut.48.3.297.
7
Inactivating mutations and overexpression of BCL10, a caspase recruitment domain-containing gene, in MALT lymphoma with t(1;14)(p22;q32).在伴有t(1;14)(p22;q32)的黏膜相关淋巴组织淋巴瘤中，含半胱天冬酶募集结构域的基因BCL10的失活突变和过表达。
Nat Genet. 1999 May;22(1):63-8. doi: 10.1038/8767.
8
Bcl10 is involved in t(1;14)(p22;q32) of MALT B cell lymphoma and mutated in multiple tumor types.Bcl10参与黏膜相关淋巴组织（MALT）B细胞淋巴瘤的t(1;14)(p22;q32)，并在多种肿瘤类型中发生突变。
Cell. 1999 Jan 8;96(1):35-45. doi: 10.1016/s0092-8674(00)80957-5.
9
Regression of gastric MALT lymphoma after eradication of Helicobacter pylori is predicted by endosonographic staging. MALT Lymphoma Study Group.胃黏膜相关淋巴组织淋巴瘤在根除幽门螺杆菌后的消退可通过内镜超声分期预测。黏膜相关淋巴组织淋巴瘤研究组。
Gastroenterology. 1997 Oct;113(4):1087-90. doi: 10.1053/gast.1997.v113.pm9322502.

Strong BCL10 nuclear expression identifies gastric MALT lymphomas that do not respond to H pylori eradication.

作者信息

Ye H, Gong L, Liu H, Ruskone-Fourmestraux A, de Jong D, Pileri S, Thiede C, Lavergne A, Boot H, Caletti G, Wündisch T, Molina T, Taal B G, Elena S, Neubauer A, Maclennan K A, Siebert R, Remstein E D, Dogan A, Du M-Q

出版信息

Gut. 2006 Jan;55(1):137-8. doi: 10.1136/gut.2005.081117.

DOI:10.1136/gut.2005.081117
PMID:16344587
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1856390/
Abstract
摘要