Dal Santo F, Ellis E, Throckmorton G S
University of Texas Southwestern Medical Center, Dallas 75235-9031.
J Oral Maxillofac Surg. 1992 Aug;50(8):791-9. doi: 10.1016/0278-2391(92)90267-4.
The masseter muscle often has been implicated as a primary cause of postreduction displacement of the fractured zygomatic complex. However, this contention has never been proved. This study compared masseter muscle force in 10 male controls with that in 10 male patients who had sustained unilateral zygomaticomaxillary complex (ZMC) fractures. Calculation of muscle force was based on measured bite force, electromyogram, and radiographic determination of muscle vectors. It was found that the masseter muscle in patients with ZMC fractures developed significantly less force than masseter muscle in controls. Following fracture, the masseter force slowly increased, but at 4 weeks following surgery the majority of patients were still well below control levels. The results of this study cast uncertainty on the role of the masseter muscle in postreduction displacement of the fractured ZMC.
咬肌常被认为是颧骨复合体骨折复位后移位的主要原因。然而,这一观点从未得到证实。本研究比较了10名男性对照组与10名单侧颧骨上颌复合体(ZMC)骨折男性患者的咬肌力量。肌肉力量的计算基于测量的咬合力、肌电图以及肌肉向量的影像学测定。结果发现,ZMC骨折患者的咬肌产生的力量明显低于对照组。骨折后,咬肌力量缓慢增加,但术后4周时,大多数患者仍远低于对照组水平。本研究结果使咬肌在ZMC骨折复位后移位中所起作用存在不确定性。