Marcer G, Bernardi G, Bartolucci G B, Mastrangelo G, Belluco U, Camposampiero A, Saia B
Institute of Occupational Medicine, University of Padua, Italy.
Br J Ind Med. 1992 Jul;49(7):489-93. doi: 10.1136/oem.49.7.489.
Two hundred and sixty seven workers employed in the manufacture of silicon carbide (SiC) were examined to determine the effects of exposure to contaminants (SiC, quartz, and SO2) in the workplace on pulmonary function. No exposure concentrations exceeded the current permissible limits. Ten subjects (3.7%) showed rounded opacities (profusion greater than or equal to 1/0). Two subjects employed only in the final stages of the production process and not exposed to crystalline silica showed opacities (profusion q1/0 and q2/1) on x ray film suggesting a role of SiC in the genesis of interstitial lung disease. Chest abnormalities on x ray film were correlated with cumulative exposure to dust and pulmonary function was affected by cumulative dust exposure, profusion of opacities, and smoking. It is concluded that the current standards do not provide adequate protection against pneumoconiosis and chronic pulmonary disease in this industry.
对267名从事碳化硅（SiC）制造的工人进行了检查，以确定工作场所接触污染物（SiC、石英和SO2）对肺功能的影响。接触浓度均未超过当前的允许限值。10名受试者（3.7%）显示有圆形阴影（密集度大于或等于1/0）。两名仅在生产过程最后阶段工作且未接触结晶二氧化硅的受试者在X光片上显示有阴影（密集度为q1/0和q2/1），提示SiC在间质性肺病的发生中起作用。X光片上的胸部异常与粉尘累积接触有关，肺功能受粉尘累积接触、阴影密集度和吸烟的影响。得出的结论是，当前标准不能为该行业预防尘肺病和慢性肺病提供充分保护。
