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Infection as a cause of arthritis.

作者信息

Moreland L W, Koopman W J

机构信息

University of Alabama, Birmingham School of Medicine.

出版信息

Curr Opin Rheumatol. 1991 Aug;3(4):639-49. doi: 10.1097/00002281-199108000-00015.

DOI:10.1097/00002281-199108000-00015
PMID:1654973
Abstract

The etiology of most rheumatic tissues is unknown; however, recent investigation into this area is rapidly providing new clues to the pathogenesis of many of these diseases. This article reviews several infectious agents (Epstein-Barr virus, parvoviruses, and mycobacteria) and the data supporting their roles in the etiology of rheumatoid arthritis. In addition, current data on the potential roles of Epstein-Barr virus and retroviruses in the etiologies of Sjögren's syndrome and Kawasaki disease are also reviewed. Finally, the reactive arthritides, for which there is stronger evidence of an infectious etiology, are reviewed.

摘要

相似文献

1
Infection as a cause of arthritis.
Curr Opin Rheumatol. 1991 Aug;3(4):639-49. doi: 10.1097/00002281-199108000-00015.
2
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Infection as a cause of reactive arthritis, ankylosing spondylitis, and rheumatic fever.感染作为反应性关节炎、强直性脊柱炎和风湿热的病因。
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引用本文的文献

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Inducible nitric oxide synthase is expressed in joints of goats in the late stage of infection with caprine arthritis encephalitis virus.诱导型一氧化氮合酶在山羊感染山羊关节炎脑炎病毒后期的关节中表达。
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Expression of cytokine mRNA in lentivirus-induced arthritis.
细胞因子mRNA在慢病毒诱导性关节炎中的表达
Am J Pathol. 1997 Oct;151(4):1053-65.
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Caprine arthritis encephalitis virus dysregulates the expression of cytokines in macrophages.山羊关节炎脑炎病毒会使巨噬细胞中细胞因子的表达失调。
J Virol. 1997 Oct;71(10):7488-97. doi: 10.1128/JVI.71.10.7488-7497.1997.
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Spontaneous establishment of an Epstein-Barr virus-infected fibroblast line from the synovial tissue of a rheumatoid arthritis patient.从一名类风湿关节炎患者的滑膜组织中自发建立爱泼斯坦-巴尔病毒感染的成纤维细胞系。
J Virol. 1997 Mar;71(3):2478-81. doi: 10.1128/JVI.71.3.2478-2481.1997.
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Rheumatoid arthritis: how well do the theories fit the evidence?类风湿性关节炎:这些理论与证据的契合程度如何?
Clin Exp Immunol. 1993 Apr;92(1):1-6. doi: 10.1111/j.1365-2249.1993.tb05938.x.
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Glycosylation of IgG during potentially arthritogenic lentiviral infections.在潜在致关节炎慢病毒感染期间IgG的糖基化
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