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钙化性神经囊尾蚴病病变在抗寄生虫治疗期间引发症状性炎症。

Calcified neurocysticercosis lesions trigger symptomatic inflammation during antiparasitic therapy.

作者信息

Poeschl P, Janzen A, Schuierer G, Winkler J, Bogdahn U, Steinbrecher A

机构信息

Department of Neurology, University of Regensburg, Regensburg, Germany.

出版信息

AJNR Am J Neuroradiol. 2006 Mar;27(3):653-5.

PMID:16552011
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7976951/
Abstract

We report a patient with neurocysticercosis who developed numerous cerebral edematous lesions while undergoing cysticidal therapy. These lesions outnumbered viable cystic lesions seen before therapy. Most new lesions were subsequently found to be associated with former calcifications not seen on initial MR imaging. Calcified neurocysticercosis lesions can trigger inflammatory reactions during therapy, and the number and location of calcified neurocysticercosis lesions may influence treatment decisions.

摘要

我们报告了一名患有神经囊尾蚴病的患者,该患者在进行杀囊治疗时出现了大量脑水肿性病变。这些病变的数量超过了治疗前可见的存活囊状病变。随后发现,大多数新病变与初始磁共振成像中未见的先前钙化灶相关。钙化性神经囊尾蚴病病变在治疗期间可引发炎症反应,钙化性神经囊尾蚴病病变的数量和位置可能会影响治疗决策。

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Clin Infect Dis. 2001 Nov 15;33(10):1649-53. doi: 10.1086/323670. Epub 2001 Oct 5.
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