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Septic shock: a heart story since the 1960s.

作者信息

Rabuel C, Mebazaa A

机构信息

University Paris 7 Denis Diderot, AP-HP, Department of Anesthesiology and Critical Care Medicine, Lariboisiere Hospital, 2 Rue Ambroise Paré, 75010 Paris, France.

出版信息

Intensive Care Med. 2006 Jun;32(6):799-807. doi: 10.1007/s00134-006-0142-5. Epub 2006 Mar 29.

DOI:10.1007/s00134-006-0142-5
PMID:16570145
Abstract
摘要

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Septic shock: a heart story since the 1960s.脓毒性休克：自20世纪60年代以来的心脏相关情况。
Intensive Care Med. 2006 Jun;32(6):799-807. doi: 10.1007/s00134-006-0142-5. Epub 2006 Mar 29.
2
The hemodynamics of septic shock: a historical perspective.脓毒性休克的血液动力学：历史视角。
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3
Mechanisms of vascular hyporesponsiveness in septic shock.感染性休克中血管低反应性的机制。
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4
Old unhappy far off things. Some reflections on the significance of the early work on shock.往昔不愉快的遥远之事。关于休克早期研究意义的一些思考。
Ann R Coll Surg Engl. 1967 May;40(5):289-305.
5
The bacterial factor in hemorrhagic shock.出血性休克中的细菌因素。
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Toxic shock syndrome--United States. 1980.中毒性休克综合征——美国，1980年。
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7
Right ventricular performance in sepsis and septic shock.脓毒症和脓毒性休克时的右心室功能
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8
Pathophysiological correlates of cardiac overperformance in sepsis and septic shock.
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9
Right ventricular dysfunction in human septic shock.
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Cardiac function in septic shock.感染性休克中的心脏功能
Ann Intern Med. 1984 Dec;101(6):879. doi: 10.7326/0003-4819-101-6-879_1.

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Cardiac and renal effects of levosimendan, arginine vasopressin, and norepinephrine in lipopolysaccharide-treated rabbits.左西孟旦、精氨酸加压素和去甲肾上腺素对脂多糖处理兔的心脏和肾脏影响
Anesthesiology. 2005 Sep;103(3):514-21. doi: 10.1097/00000542-200509000-00014.
2
Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction.心室肌细胞半胱天冬酶直接导致内毒素诱导的心脏功能障碍。
Circulation. 2005 May 24;111(20):2596-604. doi: 10.1161/CIRCULATIONAHA.104.490979. Epub 2005 May 16.
3
Protection against endotoxemia-induced contractile dysfunction in mice with cardiac-specific expression of slow skeletal troponin I.
基于夏普利加性解释的因子分析在使用XGBoost预测ICU中脓毒症相关性脑病死亡率中的应用——一项基于两个大型数据库的回顾性研究
Front Neurol. 2023 Dec 14;14:1290117. doi: 10.3389/fneur.2023.1290117. eCollection 2023.
4
Current insight on the mechanisms of programmed cell death in sepsis-induced myocardial dysfunction.脓毒症诱导心肌功能障碍中程序性细胞死亡机制的当前见解。
Front Cell Dev Biol. 2023 Dec 15;11:1309719. doi: 10.3389/fcell.2023.1309719. eCollection 2023.
5
Sepsis-induced cardiomyopathy in animals: From experimental studies to echocardiography-based clinical research.动物脓毒症性心肌病：从实验研究到基于超声心动图的临床研究。
Can Vet J. 2023 Sep;64(9):871-877.
6
Echocardiographic profiles and hemodynamic response after vasopressin initiation in septic shock: A cross-sectional study.超声心动图特征与血管加压素起始后对感染性休克血流动力学的反应：一项横断面研究。
J Crit Care. 2023 Aug;76:154298. doi: 10.1016/j.jcrc.2023.154298. Epub 2023 Apr 6.
7
Combined Multiomics and In Silico Approach Uncovers PRKAR1A as a Putative Therapeutic Target in Multi-Organ Dysfunction Syndrome.联合多组学和计算机模拟方法揭示PRKAR1A作为多器官功能障碍综合征的潜在治疗靶点。
ACS Omega. 2023 Mar 1;8(10):9555-9568. doi: 10.1021/acsomega.3c00020. eCollection 2023 Mar 14.
8
The Overexpression of miR-377 Aggravates Sepsis-Induced Myocardial Hypertrophy by Binding to Rcan2 and Mediating CaN Activity.miR-377 的过表达通过与 Rcan2 结合并介导 CaN 活性加重脓毒症诱导的心肌肥厚。
Oxid Med Cell Longev. 2022 Oct 11;2022:6659183. doi: 10.1155/2022/6659183. eCollection 2022.
9
Sepsis-Associated Encephalopathy and Blood-Brain Barrier Dysfunction.脓毒症相关性脑病与血脑屏障功能障碍
Inflammation. 2021 Dec;44(6):2143-2150. doi: 10.1007/s10753-021-01501-3. Epub 2021 Jul 21.
10
Aldehyde Dehydrogenase 2 Protects Against Lipopolysaccharide-Induced Myocardial Injury by Suppressing Mitophagy.乙醛脱氢酶2通过抑制线粒体自噬保护免受脂多糖诱导的心肌损伤。
Front Pharmacol. 2021 May 7;12:641058. doi: 10.3389/fphar.2021.641058. eCollection 2021.
在心脏特异性表达慢肌肌钙蛋白I的小鼠中预防内毒素血症诱导的收缩功能障碍。
FASEB J. 2005 Jul;19(9):1137-9. doi: 10.1096/fj.04-2519fje. Epub 2005 Apr 26.
4
Effects of levosimendan on systemic and regional hemodynamics in septic myocardial depression.左西孟旦对脓毒症性心肌抑制时全身和局部血流动力学的影响。
Intensive Care Med. 2005 May;31(5):638-44. doi: 10.1007/s00134-005-2619-z. Epub 2005 Apr 6.
5
Expression of apoptosis regulatory factors during myocardial dysfunction in endotoxemic rats.
Crit Care Med. 2005 Mar;33(3):492-6. doi: 10.1097/01.ccm.0000156240.31913.4a.
6
Peroxynitrite decomposition catalysts prevent myocardial dysfunction and inflammation in endotoxemic rats.过氧亚硝酸盐分解催化剂可预防内毒素血症大鼠的心肌功能障碍和炎症。
J Am Coll Cardiol. 2004 Jun 16;43(12):2348-58. doi: 10.1016/j.jacc.2004.01.047.
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Bacteremia due to gram-negative bacilli other than the Salmonella; a clinical and therapeutic study.除沙门氏菌外的革兰氏阴性杆菌所致菌血症：一项临床与治疗研究
AMA Arch Intern Med. 1951 Oct;88(4):467-88. doi: 10.1001/archinte.1951.03810100051005.
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Acute right ventricular failure--from pathophysiology to new treatments.急性右心室衰竭——从病理生理学到新的治疗方法
Intensive Care Med. 2004 Feb;30(2):185-196. doi: 10.1007/s00134-003-2025-3. Epub 2003 Nov 15.
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Studies on the circulatory changes in the dog produced by endotoxin from gram-negative microorganisms.革兰氏阴性微生物内毒素所致犬循环系统变化的研究。
J Clin Invest. 1956 Nov;35(11):1191-8. doi: 10.1172/JCI103373.
10
Association between mitochondrial dysfunction and severity and outcome of septic shock.线粒体功能障碍与感染性休克的严重程度及预后之间的关联。
Lancet. 2002 Jul 20;360(9328):219-23. doi: 10.1016/S0140-6736(02)09459-X.