IMMUNOREACTIONS INVOLVING PLATELETS. V. POST-TRANSFUSION PURPURA DUE TO A COMPLEMENT-FIXING ANTIBODY AGAINST A GENETICALLY CONTROLLED PLATELET ANTIGEN. A PROPOSED MECHANISM FOR THROMBOCYTOPENIA AND ITS RELEVANCE IN "AUTOIMMUNITY".
作者信息
Shulman N R, Aster R H, Leitner A, Hiller M C
机构信息
National Institute of Arthritis and Metabolic Diseases, Bethesda, Md.
出版信息
J Clin Invest. 1961 Sep;40(9):1597-620. doi: 10.1172/JCI104383.
Abstract
摘要
相似文献
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IMMUNOREACTIONS INVOLVING PLATELETS. V. POST-TRANSFUSION PURPURA DUE TO A COMPLEMENT-FIXING ANTIBODY AGAINST A GENETICALLY CONTROLLED PLATELET ANTIGEN. A PROPOSED MECHANISM FOR THROMBOCYTOPENIA AND ITS RELEVANCE IN "AUTOIMMUNITY".涉及血小板的免疫反应。五、由针对遗传控制的血小板抗原的补体结合抗体引起的输血后紫癜。血小板减少症的一种推测机制及其在“自身免疫”中的相关性。
J Clin Invest. 1961 Sep;40(9):1597-620. doi: 10.1172/JCI104383.
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Immunoreactions involving platelets. III. Quantitative aspects of platelet agglutination, inhibition of clot retraction, and other reactions caused by the antibody of quinidine purpura.涉及血小板的免疫反应。III. 血小板凝集、凝块回缩抑制及奎尼丁紫癜抗体引起的其他反应的定量研究
J Exp Med. 1958 May 1;107(5):697-710. doi: 10.1084/jem.107.5.697.
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Immunoreactions involving platelets. IV. Studies on the pathogenesis of thrombocytopenia in drug purpura using test doses of quinidine in sensitized individuals; their implications in idiopathic thrombocytopenic purpura.涉及血小板的免疫反应。IV. 使用奎尼丁试验剂量对致敏个体药物性紫癜中血小板减少症发病机制的研究；其对特发性血小板减少性紫癜的意义。
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Increased fluidity of human platelet membranes during complement-mediated immune platelet injury.补体介导的免疫性血小板损伤过程中人类血小板膜流动性增加。
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Immunoreactions involving platelets. I. A steric and kinetic model for formation of a complex from a human antibody, quinidine as a haptene, and platelets; and for fixation of complement by the complex.涉及血小板的免疫反应。I. 关于人抗体、作为半抗原的奎尼丁与血小板形成复合物以及该复合物固定补体的空间和动力学模型。
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Immunoreactions involving platelet. VI. Reactions of maternal isoantibodies responsible for neonatal purpura. Differentiation of a second platelet antigen system.涉及血小板的免疫反应。VI. 导致新生儿紫癜的母体同种抗体的反应。第二种血小板抗原系统的鉴别。
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Immunoreactions involving platelets. II. Theoretical analysis of the model.涉及血小板的免疫反应。II. 模型的理论分析。
J Exp Med. 1958 May 1;107(5):691-5. doi: 10.1084/jem.107.5.691.
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Clinical significance of platelet alloantibodies detected in immunofluorescence test (neonatal alloimmune thrombocytopenia and post-transfusion purpura).免疫荧光试验中检测到的血小板同种抗体的临床意义（新生儿同种免疫性血小板减少症和输血后紫癜）
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Post-transfusion purpura and isoimmune neonatal thrombocytopenia in the same family.同一家族中的输血后紫癜和同种免疫性新生儿血小板减少症。
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CHANGE IN PLATELET FACTOR 3 AS A MEANS OF DEMONSTRATING IMMUNE REACTIONS INVOLVING PLATELETS: ITS USE AS A TEST FOR QUINIDINE-INDUCED THROMBOCYTOPENIA.血小板第3因子的变化作为一种证明涉及血小板的免疫反应的手段：其作为奎尼丁诱导的血小板减少症检测方法的应用。
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本文引用的文献
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Acquisition of the J Substance by the Bovine Erythrocyte.牛红细胞对J物质的摄取。
Proc Natl Acad Sci U S A. 1949 May;35(5):232-7. doi: 10.1073/pnas.35.5.232.
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The cause of thrombocytopenia in sedormid purpura.
Clin Sci. 1949 Dec 19;8(4):269-89, incl pl.
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Morphology and enumeration of human blood platelets.人体血小板的形态学与计数
J Appl Physiol. 1950 Dec;3(6):365-77. doi: 10.1152/jappl.1950.3.6.365.
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The application of fluorescent antibody methods to the study of platelets.荧光抗体方法在血小板研究中的应用。
Blood. 1960 Jul;16:958-67.
5
Specific inhibition of antibody formation during immunological paralysis and unresponsiveness.免疫麻痹和无反应性期间抗体形成的特异性抑制。
Nature. 1959 Oct 3;184(Suppl 14):1080-2. doi: 10.1038/1841080a0.
6
Relationship between RhO(D) genotype and quantity of 1131 anti-RhO(D) bound to red cells.RhO(D)基因型与结合在红细胞上的1131抗RhO(D)数量之间的关系。
J Clin Invest. 1960 Sep;39(9):1450-62. doi: 10.1172/JCI104164.
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Congenital thrombocytopenic purpura.先天性血小板减少性紫癜
Arch Dis Child. 1960 Apr;35(180):146-52. doi: 10.1136/adc.35.180.146.
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Leukemia and auto-immunization- some possible relationships.白血病与自身免疫——一些可能的关系。
Blood. 1959 Oct;14:1151-8.
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