The egress of herpesviruses from cells: the unanswered questions.
作者信息
Campadelli-Fiume Gabriella, Roizman Bernard
出版信息
J Virol. 2006 Jul;80(13):6716-7; author replies 6717-9. doi: 10.1128/JVI.00386-06.
Abstract
摘要
相似文献
1
The egress of herpesviruses from cells: the unanswered questions.疱疹病毒从细胞中的释放：未解决的问题。
J Virol. 2006 Jul;80(13):6716-7; author replies 6717-9. doi: 10.1128/JVI.00386-06.
2
Egress of alphaherpesviruses.甲型疱疹病毒的释放
J Virol. 2006 Feb;80(3):1610-1; author reply 1611-2. doi: 10.1128/JVI.80.3.1610-1612.2006.
3
Herpesvirus assembly and egress.疱疹病毒的组装与释放。
J Virol. 2002 Feb;76(4):1537-47. doi: 10.1128/jvi.76.4.1537-1547.2002.
4
Herpesvirus assembly: a tale of two membranes.疱疹病毒组装：双膜的故事。
Curr Opin Microbiol. 2006 Aug;9(4):423-9. doi: 10.1016/j.mib.2006.06.013. Epub 2006 Jun 30.
5
The human cytomegalovirus assembly compartment: a masterpiece of viral manipulation of cellular processes that facilitates assembly and egress.人类巨细胞病毒组装区室：病毒对细胞过程进行操纵的杰作，有助于病毒组装和释放。
PLoS Pathog. 2012 Sep;8(9):e1002878. doi: 10.1371/journal.ppat.1002878. Epub 2012 Sep 20.
6
Structural changes in human cytomegalovirus cytoplasmic assembly sites in the absence of UL97 kinase activity.在缺乏UL97激酶活性的情况下人巨细胞病毒细胞质装配位点的结构变化
Virology. 2006 Oct 10;354(1):69-79. doi: 10.1016/j.virol.2006.05.037. Epub 2006 Jul 26.
7
Actin and Rho GTPases in herpesvirus biology.肌动蛋白和Rho GTP酶在疱疹病毒生物学中的作用
Trends Microbiol. 2007 Sep;15(9):426-33. doi: 10.1016/j.tim.2007.08.003. Epub 2007 Aug 30.
8
Three-dimensional structure of the human cytomegalovirus cytoplasmic virion assembly complex includes a reoriented secretory apparatus.人类巨细胞病毒细胞质病毒体组装复合体的三维结构包括重新定向的分泌装置。
J Virol. 2007 Nov;81(21):11861-9. doi: 10.1128/JVI.01077-07. Epub 2007 Aug 22.
9
Herpesviruses in asymptomatic apical periodontitis lesions: an immunohistochemical approach.无症状性根尖周炎病变中的疱疹病毒：一种免疫组织化学方法。
Oral Microbiol Immunol. 2007 Oct;22(5):320-5. doi: 10.1111/j.1399-302X.2007.00362.x.
10
[Morphology of human alpha-herpesviruses].[人类α疱疹病毒的形态学]
Nihon Rinsho. 2006 Mar;64 Suppl 3:121-6.
引用本文的文献
1
The Interplay between Adeno-Associated Virus and its Helper Viruses.腺相关病毒与其辅助病毒之间的相互作用。
Viruses. 2020 Jun 19;12(6):662. doi: 10.3390/v12060662.
2
Assembly and Egress of an Alphaherpesvirus Clockwork.甲型疱疹病毒“发条装置”的组装与释放
Adv Anat Embryol Cell Biol. 2017;223:171-193. doi: 10.1007/978-3-319-53168-7_8.
3
Maturation and vesicle-mediated egress of primate gammaherpesvirus rhesus monkey rhadinovirus require inner tegument protein ORF52.灵长类γ疱疹病毒恒河猴疱疹病毒的成熟和囊泡介导的排出需要内膜蛋白 ORF52。
J Virol. 2014 Aug;88(16):9111-28. doi: 10.1128/JVI.01502-14. Epub 2014 Jun 4.
4
Rab6 dependent post-Golgi trafficking of HSV1 envelope proteins to sites of virus envelopment.Rab6 依赖的 HSV1 包膜蛋白的高尔基后转运至病毒包膜形成部位。
Traffic. 2014 Feb;15(2):157-78. doi: 10.1111/tra.12134. Epub 2013 Nov 18.
5
Polarized cell migration during cell-to-cell transmission of herpes simplex virus in human skin keratinocytes.单纯疱疹病毒在人皮肤角质细胞间传递时细胞的极化迁移。
J Virol. 2013 Jul;87(14):7921-32. doi: 10.1128/JVI.01172-13. Epub 2013 May 8.
6
Packaging of Non-Coding RNAs into Herpesvirus Virions: Comparisons to Coding RNAs.非编码RNA包装进疱疹病毒病毒粒子：与编码RNA的比较
Front Genet. 2011 Nov 17;2:81. doi: 10.3389/fgene.2011.00081. eCollection 2011.
7
Alphaherpesviruses and the cytoskeleton in neuronal infections.α疱疹病毒与神经元感染中的细胞骨架。
Viruses. 2011 Jul;3(7):941-81. doi: 10.3390/v3070941. Epub 2011 Jun 27.
8
Ultrastructural analysis of ICP34.5- herpes simplex virus 1 replication in mouse brain cells in vivo.体内 ICP34.5-单纯疱疹病毒 1 在小鼠脑细胞中的复制的超微结构分析。
J Virol. 2010 Nov;84(21):10982-90. doi: 10.1128/JVI.00337-10. Epub 2010 Aug 11.
9
Nucleolin is required for efficient nuclear egress of herpes simplex virus type 1 nucleocapsids.核仁蛋白对于单纯疱疹病毒 1 核衣壳的有效核输出是必需的。
J Virol. 2010 Feb;84(4):2110-21. doi: 10.1128/JVI.02007-09. Epub 2009 Dec 2.
10
Nuclear pore composition and gating in herpes simplex virus-infected cells.单纯疱疹病毒感染细胞中的核孔组成与门控
J Virol. 2008 Sep;82(17):8392-9. doi: 10.1128/JVI.00951-08. Epub 2008 Jun 18.
本文引用的文献
1
Herpes simplex virus tegument protein VP16 is a component of primary enveloped virions.单纯疱疹病毒被膜蛋白VP16是初级包膜病毒体的一个组成部分。
J Virol. 2006 Mar;80(5):2582-4. doi: 10.1128/JVI.80.5.2582-2584.2006.
2
Egress of alphaherpesviruses.甲型疱疹病毒的释放
J Virol. 2006 Feb;80(3):1610-1; author reply 1611-2. doi: 10.1128/JVI.80.3.1610-1612.2006.
3
Herpes simplex virus 1 envelopment follows two diverse pathways.单纯疱疹病毒1型的包膜形成遵循两条不同的途径。
J Virol. 2005 Oct;79(20):13047-59. doi: 10.1128/JVI.79.20.13047-13059.2005.
4
The ectodomain of herpes simplex virus glycoprotein H contains a membrane alpha-helix with attributes of an internal fusion peptide, positionally conserved in the herpesviridae family.单纯疱疹病毒糖蛋白H的胞外结构域包含一个具有内部融合肽特性的膜α螺旋，在疱疹病毒科中位置保守。
J Virol. 2005 Mar;79(5):2931-40. doi: 10.1128/JVI.79.5.2931-2940.2005.
5
Impairment of nuclear pores in bovine herpesvirus 1-infected MDBK cells.牛疱疹病毒1感染的MDBK细胞中核孔的损伤
J Virol. 2005 Jan;79(2):1071-83. doi: 10.1128/JVI.79.2.1071-1083.2005.
6
Herpes simplex virus type 1 gK is required for gB-mediated virus-induced cell fusion, while neither gB and gK nor gB and UL20p function redundantly in virion de-envelopment.单纯疱疹病毒1型gK是gB介导的病毒诱导细胞融合所必需的，而在病毒脱壳过程中，gB与gK之间以及gB与UL20p之间均不存在功能冗余。
J Virol. 2005 Jan;79(1):299-313. doi: 10.1128/JVI.79.1.299-313.2005.
7
Budding events in herpesvirus morphogenesis.疱疹病毒形态发生中的出芽事件。
Virus Res. 2004 Dec;106(2):167-80. doi: 10.1016/j.virusres.2004.08.013.
8
Coexpression of UL20p and gK inhibits cell-cell fusion mediated by herpes simplex virus glycoproteins gD, gH-gL, and wild-type gB or an endocytosis-defective gB mutant and downmodulates their cell surface expression.UL20p和gK的共表达可抑制单纯疱疹病毒糖蛋白gD、gH-gL以及野生型gB或内吞作用缺陷型gB突变体介导的细胞间融合，并下调它们在细胞表面的表达。
J Virol. 2004 Aug;78(15):8015-25. doi: 10.1128/JVI.78.15.8015-8025.2004.
9
Simultaneous deletion of pseudorabies virus tegument protein UL11 and glycoprotein M severely impairs secondary envelopment.伪狂犬病病毒被膜蛋白UL11和糖蛋白M的同时缺失严重损害了二次包膜化过程。
J Virol. 2004 Mar;78(6):3024-34. doi: 10.1128/jvi.78.6.3024-3034.2004.
10
Envelopment of human cytomegalovirus occurs by budding into Golgi-derived vacuole compartments positive for gB, Rab 3, trans-golgi network 46, and mannosidase II.人巨细胞病毒的包膜形成是通过出芽进入高尔基体衍生的、对gB、Rab 3、反式高尔基体网络46和甘露糖苷酶II呈阳性的液泡区室来实现的。
J Virol. 2003 Mar;77(5):3191-203. doi: 10.1128/jvi.77.5.3191-3203.2003.
Zotero 插件