可逆性迟发性缺氧后白质脑病
Reversible delayed posthypoxic leukoencephalopathy.
作者信息
Molloy S, Soh C, Williams T L
机构信息
Department of Neurology, Royal Victoria Infirmary, Newcastle upon Tyne, NE1 4LP, England.
出版信息
AJNR Am J Neuroradiol. 2006 Sep;27(8):1763-5.
Abstract
Hypoxic ischemic encephalopathy may cause early deep white matter abnormalities on MR imaging that usually progress to include gray matter and basal ganglia change. Toxic leukoencephalopathy due to heroin inhalation predominantly causes cerebellar and posterior cerebral radiologic change. Both conditions rarely present clinically and radiologically in a delayed manner with subsequent recovery. We report a case of reversible delayed posthypoxic/toxic leukoencephalopathy with no clinical or radiologic evidence of gray matter insult.
摘要
缺氧缺血性脑病可能在磁共振成像上导致早期深部白质异常，这些异常通常会进展至包括灰质和基底节改变。因吸入海洛因所致的中毒性白质脑病主要引起小脑和大脑后部的影像学改变。这两种情况临床上和影像学上都很少以延迟方式出现并随后恢复。我们报告一例可逆性延迟性缺氧/中毒性白质脑病病例，该病例无灰质损伤的临床或影像学证据。
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