Bouzgarou E, Dupeyron A, Castelnovo G, Boudousq V, Collombier L, Labauge P, Pélissier J
Département de MPR, groupe hospitalier Carémeau, 30029 Nîmes cedex 04, France.
Ann Readapt Med Phys. 2007 Jan;50(1):55-9. doi: 10.1016/j.annrmp.2006.07.060. Epub 2006 Sep 1.
Camptocormia is characterised as an extreme bent-forward posture of the trunk that disappears in the recumbent position. On X-ray, trunk flexion appears without vertebral rotation as in scoliosis. The condition is a well-known complication of Parkinson's disease (PD) at the late stage. The authors present the case of a 77-year-old woman affected by severe camptocormia, which appeared and worsened in less than 6 months and hindered gait. Despite no signs of PD, neuro-imaging (DAT-Scan) showed an L-Dopa transducer decrease in putamens. A few weeks later, bradykinesia appeared and the clinical diagnosis of PD became more obvious. L-Dopa improved bradykinesia but did not change the bent-spine posture. A 1-year follow-up showed no other signs of PD other than bradykinesia, but the camptocormia was unchanged.
camptocormia的特征是躯干极度向前弯曲的姿势，在卧位时消失。在X射线下，躯干前屈出现但无脊柱侧弯时的椎体旋转。这种情况是帕金森病（PD）晚期的一种众所周知的并发症。作者介绍了一名77岁严重camptocormia女性的病例，该症状在不到6个月的时间内出现并恶化，妨碍了步态。尽管没有帕金森病的迹象，但神经影像学检查（DAT扫描）显示壳核中左旋多巴转导器减少。几周后，出现运动迟缓，帕金森病的临床诊断变得更加明显。左旋多巴改善了运动迟缓，但没有改变脊柱弯曲的姿势。1年的随访显示，除运动迟缓外没有其他帕金森病的迹象，但camptocormia没有变化。
