65kD热休克蛋白：介导自身免疫性关节炎发展的关键分子？ - Suppr

Suppr

超能文献

The 65kD heat shock protein: a key molecule mediating the development of autoimmune arthritis?

作者信息

Yang X D, Feige U

机构信息

Research Department, Ciba-Geigy Ltd., Basle, Switzerland.

出版信息

Autoimmunity. 1991;9(1):83-8. doi: 10.3109/08916939108997128.

DOI:10.3109/08916939108997128

PMID:1727019

Abstract

摘要

相似文献

The 65kD heat shock protein: a key molecule mediating the development of autoimmune arthritis?65kD热休克蛋白：介导自身免疫性关节炎发展的关键分子？

Autoimmunity. 1991;9(1):83-8. doi: 10.3109/08916939108997128.

Antibody reactivity to mycobacterial 65 kDa heat shock protein: relevance to autoimmunity.抗分枝杆菌65 kDa热休克蛋白的抗体反应性：与自身免疫的相关性。

J Autoimmun. 1995 Apr;8(2):235-48. doi: 10.1006/jaut.1995.0018.

Heat shock protein 65-reactive T cells are involved in the pathogenesis of non-antigenic dimethyl dioctadecyl ammonium bromide-induced arthritis.热休克蛋白65反应性T细胞参与了非抗原性二甲基二十八烷基溴化铵诱导的关节炎的发病机制。

J Immunol. 2005 Jul 1;175(1):219-27. doi: 10.4049/jimmunol.175.1.219.

Diversification of T cell responses to carboxy-terminal determinants within the 65-kD heat-shock protein is involved in regulation of autoimmune arthritis.T细胞对65-kD热休克蛋白羧基末端决定簇反应的多样化参与自身免疫性关节炎的调控。

J Exp Med. 1997 Apr 7;185(7):1307-16. doi: 10.1084/jem.185.7.1307.

T cells against the pathogenic and protective epitopes of heat-shock protein 65 are crossreactive and display functional similarity: novel aspect of regulation of autoimmune arthritis.针对热休克蛋白65致病性和保护性表位的T细胞具有交叉反应性并表现出功能相似性：自身免疫性关节炎调节的新方面

J Rheumatol. 2007 Nov;34(11):2134-43. Epub 2007 Oct 15.

Regulation of autoimmune arthritis by self-heat-shock proteins.自身热休克蛋白对自身免疫性关节炎的调节作用。

Trends Immunol. 2008 Sep;29(9):412-8. doi: 10.1016/j.it.2008.06.003.

Differential T-cell recognition of cathepsin D-released mycobacterial Hsp65 fragments by arthritic versus healthy Lewis rats.关节炎Lewis大鼠与健康Lewis大鼠对组织蛋白酶D释放的分枝杆菌热休克蛋白65片段的差异性T细胞识别

Transplant Proc. 1993 Oct;25(5):2840-1.

Resistance to adjuvant arthritis is due to protective antibodies against heat shock protein surface epitopes and the induction of IL-10 secretion.对佐剂性关节炎的抗性归因于针对热休克蛋白表面表位的保护性抗体以及白细胞介素-10分泌的诱导。

J Immunol. 2002 Jun 15;168(12):6463-9. doi: 10.4049/jimmunol.168.12.6463.

The regulatory C-terminal determinants within mycobacterial heat shock protein 65 are cryptic and cross-reactive with the dominant self homologs: implications for the pathogenesis of autoimmune arthritis.分枝杆菌热休克蛋白65内的调节性C末端决定簇是隐蔽的，且与主要的自身同源物发生交叉反应：对自身免疫性关节炎发病机制的影响

J Immunol. 2004 Jul 1;173(1):181-8. doi: 10.4049/jimmunol.173.1.181.

Heat shock proteins and arthritis.热休克蛋白与关节炎

Bull Rheum Dis. 1992;41(3):4-5.

引用本文的文献

The 65-kDa heat-shock protein in the pathogenesis, prevention and therapy of autoimmune arthritis and diabetes mellitus in rats and mice.65千道尔顿热休克蛋白在大鼠和小鼠自身免疫性关节炎及糖尿病发病机制、预防和治疗中的作用

Springer Semin Immunopathol. 1991;13(1):99-113. doi: 10.1007/BF01225281.

Heat shock proteins in autoimmune disease. From causative antigen to specific therapy?自身免疫性疾病中的热休克蛋白。从致病抗原到特异性治疗？

Experientia. 1992 Jul 15;48(7):650-6. doi: 10.1007/BF02118311.

Prevention of adjuvant arthritis in rats by a nonapeptide from the 65-kD mycobacterial heat shock protein: specificity and mechanism.来自65-kD分枝杆菌热休克蛋白的九肽对大鼠佐剂性关节炎的预防作用：特异性及机制

Clin Exp Immunol. 1992 Jan;87(1):99-104. doi: 10.1111/j.1365-2249.1992.tb06420.x.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验