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The 65-kDa heat-shock protein in the pathogenesis, prevention and therapy of autoimmune arthritis and diabetes mellitus in rats and mice.

作者信息

Feige U, Cohen I R

机构信息

Department of Inflammation, Ciba-Geigy Ltd., Basle, Switzerland.

出版信息

Springer Semin Immunopathol. 1991;13(1):99-113. doi: 10.1007/BF01225281.

DOI:10.1007/BF01225281
PMID:1776121
Abstract
摘要

相似文献

1
The 65-kDa heat-shock protein in the pathogenesis, prevention and therapy of autoimmune arthritis and diabetes mellitus in rats and mice.65千道尔顿热休克蛋白在大鼠和小鼠自身免疫性关节炎及糖尿病发病机制、预防和治疗中的作用
Springer Semin Immunopathol. 1991;13(1):99-113. doi: 10.1007/BF01225281.
2
Induction and therapy of autoimmune diabetes in the non-obese diabetic (NOD/Lt) mouse by a 65-kDa heat shock protein.65 kDa热休克蛋白诱导非肥胖糖尿病（NOD/Lt）小鼠自身免疫性糖尿病及相关治疗
Proc Natl Acad Sci U S A. 1990 Feb;87(4):1576-80. doi: 10.1073/pnas.87.4.1576.
3
The 65kD heat shock protein: a key molecule mediating the development of autoimmune arthritis?65kD热休克蛋白：介导自身免疫性关节炎发展的关键分子？
Autoimmunity. 1991;9(1):83-8. doi: 10.3109/08916939108997128.
4
Heat shock proteins in autoimmune disease. From causative antigen to specific therapy?自身免疫性疾病中的热休克蛋白。从致病抗原到特异性治疗？
Experientia. 1992 Jul 15;48(7):650-6. doi: 10.1007/BF02118311.
5
Autoimmune endocrine disease.自身免疫性内分泌疾病
Vitam Horm. 1985;42:253-314. doi: 10.1016/s0083-6729(08)60064-3.
6
Vaccination against autoimmune mouse diabetes with a T-cell epitope of the human 65-kDa heat shock protein.用人65 kDa热休克蛋白的T细胞表位对自身免疫性小鼠糖尿病进行疫苗接种。
Proc Natl Acad Sci U S A. 1991 Apr 15;88(8):3088-91. doi: 10.1073/pnas.88.8.3088.
7
Early intervention in insulin-dependent diabetes mellitus.胰岛素依赖型糖尿病的早期干预
Curr Probl Clin Biochem. 1983;12:108-18.
8
Approaches to prevention and treatment of IDDM in animal models.动物模型中胰岛素依赖型糖尿病（IDDM）的预防和治疗方法。
Diabetes Care. 1988 Nov-Dec;11 Suppl 1:29-36.
9
Prevention of diabetes in NOD mice treated with antibody to murine IFN gamma.用抗小鼠γ干扰素抗体治疗的非肥胖糖尿病（NOD）小鼠的糖尿病预防
J Autoimmun. 1991 Apr;4(2):237-48. doi: 10.1016/0896-8411(91)90021-4.
10
Heat shock proteins: immunity and immunopathology.热休克蛋白：免疫与免疫病理学
Immunol Today. 1990 Jul;11(7):228-9. doi: 10.1016/0167-5699(90)90091-m.

引用本文的文献

1
HSP60: issues and insights on its therapeutic use as an immunoregulatory agent.热休克蛋白 60：作为免疫调节剂的治疗用途的问题与展望。
Front Immunol. 2012 Jan 12;2:97. doi: 10.3389/fimmu.2011.00097. eCollection 2011.
2
Hsp70--a multi-gene, multi-structure, multi-function family with potential clinical applications.热休克蛋白70——一个具有潜在临床应用价值的多基因、多结构、多功能家族。
Experientia. 1994 Nov 30;50(11-12):979-86. doi: 10.1007/BF01923452.
3
Presence of hsp65 in bacterial extracts (OM-89): a possible mediator of orally-induced tolerance?

本文引用的文献

1
PASSIVE TRANSFER OF ADJUVANT ARTHRITIS IN RATS WITH LIVING LYMPHOID CELLS OF SENSITIZED DONORS.用致敏供体的活淋巴细胞将佐剂性关节炎被动转移至大鼠体内。
Int Arch Allergy Appl Immunol. 1963;23:129-39. doi: 10.1159/000229412.
2
Adjuvant polyarthritis. IV. Induction by a synthetic adjuvant: immunologic, histopathologic, and other studies.佐剂性多关节炎。IV. 由一种合成佐剂诱导：免疫学、组织病理学及其他研究。
Arthritis Rheum. 1980 Jan;23(1):62-71. doi: 10.1002/art.1780230111.
3
Vaccination against autoimmune encephalomyelitis with T-lymphocyte line cells reactive against myelin basic protein.
细菌提取物（OM-89）中热休克蛋白65的存在：口服诱导耐受性的可能介质？
Experientia. 1995 Aug 16;51(8):775-9. doi: 10.1007/BF01922429.
4
Hsp70: a carrier molecule with built-in adjuvanticity.热休克蛋白70：一种具有内在佐剂活性的载体分子。
Experientia. 1994 Nov 30;50(11-12):1061-6. doi: 10.1007/BF01923462.
5
Heat shock proteins in autoimmune disease. From causative antigen to specific therapy?自身免疫性疾病中的热休克蛋白。从致病抗原到特异性治疗？
Experientia. 1992 Jul 15;48(7):650-6. doi: 10.1007/BF02118311.
6
Heat shock proteins. Introduction.热休克蛋白。引言。
Experientia. 1992 Jul 15;48(7):621-2. doi: 10.1007/BF02118305.
用针对髓鞘碱性蛋白的T淋巴细胞系细胞对自身免疫性脑脊髓炎进行疫苗接种。
Nature. 1981 Jul 2;292(5818):60-1. doi: 10.1038/292060a0.
4
Adjuvant polyarthritis. VIII. Differences in immunopathogenesis between type II collagen arthritis and adjuvant arthritis.佐剂性多关节炎。VIII. II型胶原性关节炎与佐剂性关节炎免疫发病机制的差异。
Agents Actions. 1984 Dec;15(5-6):529-34. doi: 10.1007/BF01966768.
5
Lines of T lymphocytes induce or vaccinate against autoimmune arthritis.T淋巴细胞系可诱导针对自身免疫性关节炎的免疫反应或进行疫苗接种。
Science. 1983 Jan 7;219(4580):56-8. doi: 10.1126/science.6336851.
6
Arthritis induced in rats by cloned T lymphocytes responsive to mycobacteria but not to collagen type II.由对分枝杆菌有反应但对II型胶原无反应的克隆T淋巴细胞在大鼠中诱导产生的关节炎。
J Clin Invest. 1984 Jan;73(1):211-5. doi: 10.1172/JCI111193.
7
Models of arthritis and the search for anti-arthritic drugs.关节炎模型与抗关节炎药物的探索。
Pharmacol Ther. 1983;21(3):389-428. doi: 10.1016/0163-7258(83)90062-1.
8
Studies of the mechanism whereby adjuvant disease is suppressed in rats pretreated with mycobacteria.对用分枝杆菌预处理的大鼠中佐剂病被抑制的机制的研究。
Int Arch Allergy Appl Immunol. 1967;31(1):57-68. doi: 10.1159/000229854.
9
Arthritis induced by a T-lymphocyte clone that responds to Mycobacterium tuberculosis and to cartilage proteoglycans.由对结核分枝杆菌和软骨蛋白聚糖有反应的T淋巴细胞克隆诱导的关节炎。
Proc Natl Acad Sci U S A. 1985 Aug;82(15):5117-20. doi: 10.1073/pnas.82.15.5117.
10
T lymphocyte clones illuminate pathogenesis and affect therapy of experimental arthritis.T淋巴细胞克隆阐明了实验性关节炎的发病机制并影响其治疗。
Arthritis Rheum. 1985 Aug;28(8):841-5. doi: 10.1002/art.1780280802.