Theodossiadis P G, Grigoropoulos V G, Emfietzoglou J, Nikolaidis P, Theodossiadis G P
2nd Department of Ophthalmology, University of Athens, 23 Likiou Street, 10674 Athens, Greece.
Eur J Ophthalmol. 2007 Mar-Apr;17(2):272-6. doi: 10.1177/112067210701700221.
To evaluate by optical coherence tomography (OCT) the vitreous involvement in full thickness macular hole associated with optic disk pit maculopathy.
Two patients with optic disk pit maculopathy and full thickness macular hole underwent OCT for evaluation of vitreous involvement in the pathogenesis of the disease. Fluorescein angiography and fundus photographs were also performed.
OCT documented the presence of vitreous traction at the edge of the full thickness macular hole in the first case and on the elevated macula in the second case. Partial posterior vitreous detachment was also noticed.
Previously unreported OCT findings such as vitreous traction at the edge of the macular hole and on the elevated macula were found. OCT provided evidence for vitreous participation in full thickness macular hole associated with optic disk pit maculopathy. OCT was also able to show that the full thickness macular hole and optic disk pit maculopathy were probably caused by similar vitreous tractional forces.
通过光学相干断层扫描（OCT）评估与视盘小凹黄斑病变相关的全层黄斑裂孔中的玻璃体受累情况。
两名患有视盘小凹黄斑病变和全层黄斑裂孔的患者接受了OCT检查，以评估玻璃体在该疾病发病机制中的受累情况。还进行了荧光素血管造影和眼底照相。
OCT记录了第一例全层黄斑裂孔边缘以及第二例黄斑隆起处存在玻璃体牵拉。还注意到了部分玻璃体后脱离。
发现了以前未报告的OCT表现，如黄斑裂孔边缘和黄斑隆起处的玻璃体牵拉。OCT为玻璃体参与与视盘小凹黄斑病变相关的全层黄斑裂孔提供了证据。OCT还能够表明全层黄斑裂孔和视盘小凹黄斑病变可能是由相似的玻璃体牵拉力引起的。
