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Calcium channels are models of self-control.

作者信息

Dunlap Kathleen

机构信息

Department of Neuroscience, Tufts University School of Medicine, Boston, MA 02111, USA.

出版信息

J Gen Physiol. 2007 May;129(5):379-83. doi: 10.1085/jgp.200709786. Epub 2007 Apr 16.

DOI:10.1085/jgp.200709786
PMID:17438121
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2154381/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6f49/2154381/a24e88f86fc7/jgp1290379f02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6f49/2154381/640d77c0d98a/jgp1290379f01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6f49/2154381/a24e88f86fc7/jgp1290379f02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6f49/2154381/640d77c0d98a/jgp1290379f01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6f49/2154381/a24e88f86fc7/jgp1290379f02.jpg

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本文引用的文献

1
Elementary mechanisms producing facilitation of Cav2.1 (P/Q-type) channels.产生Cav2.1(P/Q型)通道易化作用的基本机制。
J Gen Physiol. 2007 May;129(5):385-401. doi: 10.1085/jgp.200709749. Epub 2007 Apr 16.
2
Alternative splicing of the Ca(v)1.3 channel IQ domain, a molecular switch for Ca2+-dependent inactivation within auditory hair cells.Ca(v)1.3通道IQ结构域的可变剪接,听觉毛细胞内钙依赖性失活的分子开关。
J Neurosci. 2006 Oct 18;26(42):10690-9. doi: 10.1523/JNEUROSCI.2093-06.2006.
3
Switching of Ca2+-dependent inactivation of Ca(v)1.3 channels by calcium binding proteins of auditory hair cells.
选择性滤器门控控制电压门控钙通道钙依赖性失活。
Neuron. 2019 Mar 20;101(6):1134-1149.e3. doi: 10.1016/j.neuron.2019.01.011. Epub 2019 Feb 4.
4
Allosteric regulators selectively prevent Ca-feedback of Ca and Na channels.变构调节剂选择性地阻止钙和钠通道的钙反馈。
Elife. 2018 Sep 10;7:e35222. doi: 10.7554/eLife.35222.
5
Bilobal architecture is a requirement for calmodulin signaling to Ca1.3 channels.双叶结构是钙调蛋白信号传递到 Ca1.3 通道的必要条件。
Proc Natl Acad Sci U S A. 2018 Mar 27;115(13):E3026-E3035. doi: 10.1073/pnas.1716381115. Epub 2018 Mar 12.
6
A rendezvous with the queen of ion channels: Three decades of ion channel research by David T Yue and his Calcium Signals Laboratory.与离子通道女王的一次会面:大卫·T·岳及其钙信号实验室三十年的离子通道研究
Channels (Austin). 2016;10(1):20-32. doi: 10.1080/19336950.2015.1051272. Epub 2015 Jul 15.
7
Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels.迈向电压门控钙通道和钠通道钙调蛋白调节(钙调制)的统一理论
Curr Mol Pharmacol. 2015;8(2):188-205. doi: 10.2174/1874467208666150507110359.
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Molecular mechanism of Spinocerebellar Ataxia type 6: glutamine repeat disorder, channelopathy and transcriptional dysregulation. The multifaceted aspects of a single mutation.脊髓小脑性共济失调 6 型的分子机制:谷氨酸重复紊乱、通道病和转录失调。单一突变的多方面。
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Cell. 2014 Jun 19;157(7):1657-70. doi: 10.1016/j.cell.2014.04.035.
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J Gen Physiol. 2014 Jun;143(6):679-92. doi: 10.1085/jgp.201311153.
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J Neurosci. 2006 Oct 18;26(42):10677-89. doi: 10.1523/JNEUROSCI.3236-06.2006.
4
Structure of calmodulin bound to the hydrophobic IQ domain of the cardiac Ca(v)1.2 calcium channel.与心脏Ca(v)1.2钙通道疏水性IQ结构域结合的钙调蛋白结构
Structure. 2005 Dec;13(12):1881-6. doi: 10.1016/j.str.2005.09.021.
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Insights into voltage-gated calcium channel regulation from the structure of the CaV1.2 IQ domain-Ca2+/calmodulin complex.从CaV1.2 IQ结构域 - Ca2+/钙调蛋白复合物结构洞察电压门控钙通道调节机制
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Alternative splicing as a molecular switch for Ca2+/calmodulin-dependent facilitation of P/Q-type Ca2+ channels.可变剪接作为Ca2+/钙调蛋白依赖性促进P/Q型Ca2+通道的分子开关。
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10
Unified mechanisms of Ca2+ regulation across the Ca2+ channel family.钙离子通道家族中钙离子调节的统一机制。
Neuron. 2003 Sep 11;39(6):951-60. doi: 10.1016/s0896-6273(03)00560-9.