Dora S K, Namboodiri N, Valaparambil A K, Tharakan J A
Department of Cardiology, Sree Chitra Tirunal Institute for Medical Sciences and Technology, Thiruvananthapuram, Kerala, India.
Singapore Med J. 2007 May;48(5):e130-2.
Adenosine, used to terminate paroxysmal supraventricular tachycardia (SVT), is often useful in understanding the mechanism of tachycardia. This case report describes induction of SVT with adenosine in a 36-year-old man presenting with recurrent palpitations. After a short run of conduction via both slow and fast pathways, SVT was induced following a long PR interval. The long PR interval resulted by conduction via the slow pathway due to the preferential conduction block by adenosine over fast pathway. The notching at the terminal part of QRS during antegrade slow pathway conduction and during tachycardia indicated activation of the atrium via retrograde fast pathway. This electrocardiographical feature confirmed the mechanism of the tachycardia as atrioventricular nodal reentrant tachycardia.
用于终止阵发性室上性心动过速(SVT)的腺苷,通常有助于理解心动过速的机制。本病例报告描述了一名36岁反复心悸男性患者使用腺苷诱发SVT的情况。在经过短暂的慢径和快径传导后,在长PR间期后诱发了SVT。长PR间期是由于腺苷对快径的优先传导阻滞导致通过慢径传导所致。顺向慢径传导和心动过速期间QRS波终末部的切迹表明通过逆向快径激活了心房。这一心电图特征证实了心动过速的机制为房室结折返性心动过速。