Absence of senescence-associated beta-galactosidase activity in human melanocytic nevi in vivo.
作者信息
Cotter Murray A, Florell Scott R, Leachman Sancy A, Grossman Douglas
出版信息
J Invest Dermatol. 2007 Oct;127(10):2469-71. doi: 10.1038/sj.jid.5700903. Epub 2007 May 24.
Abstract
摘要
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Absence of senescence-associated beta-galactosidase activity in human melanocytic nevi in vivo.人黑素细胞痣在体内缺乏衰老相关的β-半乳糖苷酶活性。
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Anti-oncogenic role of the endoplasmic reticulum differentially activated by mutations in the MAPK pathway.由丝裂原活化蛋白激酶（MAPK）途径中的突变差异激活的内质网的抗癌作用。
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Oncogene-induced cell senescence--halting on the road to cancer.癌基因诱导的细胞衰老——在通往癌症的道路上停滞不前。
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Congenital melanocytic nevi frequently harbor NRAS mutations but no BRAF mutations.先天性黑素细胞痣常伴有NRAS基因突变，但无BRAF基因突变。
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Cellular senescence in naevi and immortalisation in melanoma: a role for p16?痣中的细胞衰老与黑色素瘤中的永生化：p16的作用？
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Senescence-associated beta-galactosidase is lysosomal beta-galactosidase.衰老相关β-半乳糖苷酶是溶酶体β-半乳糖苷酶。
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BRAFE600-associated senescence-like cell cycle arrest of human naevi.BRAF V600相关的人类痣细胞类似衰老的细胞周期停滞
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Apoptosis regulators and responses in human melanocytic and keratinocytic cells.人类黑素细胞和角质形成细胞中的细胞凋亡调节因子及反应
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J Cell Sci. 2000 Oct;113 ( Pt 20):3613-22. doi: 10.1242/jcs.113.20.3613.
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Am J Pathol. 2000 Sep;157(3):967-72. doi: 10.1016/S0002-9440(10)64609-3.
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