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本文引用的文献
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Myometrial expression of small conductance Ca2+-activated K+ channels depresses phasic uterine contraction.
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KCNE3 is an inhibitory subunit of the Kv4.3 potassium channel.
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Translocation of an endoproteolytically cleaved maxi-K channel isoform: mechanisms to induce human myometrial cell repolarization.
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Physiological roles of ATP-sensitive K+ channels in smooth muscle.
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Gestational change of K+ channel opener effect is correlated with the expression of uterine KATP channel subunits.
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Alternative splicing of Slo channel gene programmed by estrogen, progesterone and pregnancy.
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Functional coupling of beta3-adrenoceptors and large conductance calcium-activated potassium channels in human uterine myocytes.
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Expression of TASK and TREK, two-pore domain K+ channels, in human myometrium.
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Calcium-activated chloride channels.
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Maxi-K channels localize to caveolae in human myometrium: a role for an actin-channel-caveolin complex in the regulation of myometrial smooth muscle K+ current.
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