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Effect of diazoxide and Ca2+ on rat heart mitochondria loaded with Na+.

作者信息

Korotkov S M, Nesterov V P, Demina I N, Ryabchikov N N

机构信息

Sechenov Institute of Evolutionary Physiology and Biochemistry, Russian Academy of Sciences, pr. Morisa Toreza 44, St. Petersburg, 194223 Russia.

出版信息

Dokl Biochem Biophys. 2007 May-Jun;414:141-5. doi: 10.1134/s1607672907030131.

DOI:10.1134/s1607672907030131
PMID:17695322
Abstract
摘要

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本文引用的文献

1
Study of the mechanism of action of diazoxide on rat heart mitochondria under calcium loading.钙负荷下二氮嗪对大鼠心脏线粒体作用机制的研究。
Dokl Biochem Biophys. 2006 May-Jun;408:133-7. doi: 10.1134/s1607672906030070.
2
The direct physiological effects of mitoK(ATP) opening on heart mitochondria.线粒体ATP敏感性钾通道开放对心脏线粒体的直接生理效应。
Am J Physiol Heart Circ Physiol. 2006 Jan;290(1):H406-15. doi: 10.1152/ajpheart.00794.2005. Epub 2005 Sep 2.
3
Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.
心肌细胞中的线粒体钙激活钾通道：蛋白激酶A介导的心脏保护作用机制及调节
Circulation. 2005 Jan 18;111(2):198-203. doi: 10.1161/01.CIR.0000151099.15706.B1. Epub 2004 Dec 27.
4
Acidification reduces mitochondrial calcium uptake in rat cardiac mitochondria.
Am J Physiol Heart Circ Physiol. 2004 Dec;287(6):H2659-65. doi: 10.1152/ajpheart.00344.2004. Epub 2004 Aug 12.
5
Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration.线粒体ATP敏感性钾通道的开放通过调节线粒体基质体积、钙积累和呼吸作用增强心脏保护作用。
Am J Physiol Heart Circ Physiol. 2004 Nov;287(5):H1967-76. doi: 10.1152/ajpheart.00338.2004. Epub 2004 Jul 8.
6
Potassium channel openers are uncoupling protonophores: implication in cardioprotection.钾通道开放剂是解偶联质子载体：对心脏保护的意义。
FEBS Lett. 2004 Jun 18;568(1-3):167-70. doi: 10.1016/j.febslet.2004.05.031.
7
Diazoxide affects the IF1 inhibitor protein binding to F1 sector of beef heart F0F1ATPsynthase.二氮嗪影响IF1抑制蛋白与牛心F0F1ATP合酶F1部分的结合。
Biochem Pharmacol. 2004 May 15;67(10):1843-51. doi: 10.1016/j.bcp.2004.02.001.
8
Difference in the cardioprotective mechanisms between ischemic preconditioning and pharmacological preconditioning by diazoxide in rat hearts.大鼠心脏中缺血预处理与二氮嗪药物预处理之间心脏保护机制的差异。
Circ J. 2004 Feb;68(2):156-62. doi: 10.1253/circj.68.156.
9
Adenine nucleotide translocase mediates the K(ATP)-channel-openers-induced proton and potassium flux to the mitochondrial matrix.腺嘌呤核苷酸转位酶介导钾离子ATP通道开放剂诱导的质子和钾离子向线粒体基质的通量。
J Bioenerg Biomembr. 2003 Apr;35(2):141-8. doi: 10.1023/a:1023746103401.
10
The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respiration.缺血预处理、二氮嗪和5-羟基癸酸对大鼠心脏线粒体体积和呼吸的影响。
J Physiol. 2002 Dec 15;545(3):961-74. doi: 10.1113/jphysiol.2002.031484.