多瘤病毒非转化宿主范围突变体中组蛋白乙酰化缺陷。
Deficiency in histone acetylation in nontransforming host range mutants of polyoma virus.
作者信息
Schaffhausen B S, Benjamin T L
出版信息
Proc Natl Acad Sci U S A. 1976 Apr;73(4):1092-6. doi: 10.1073/pnas.73.4.1092.
Abstract
Histones H3 and H4 derived from transforming wild-type polyoma and simian virus 40 particles show extensive acetylation compared to the histones of the host cells. The same histone fractions derived from nontransforming host range mutants of polyoma virus fail to show this high degree of acetylation.
摘要
与宿主细胞的组蛋白相比，源自转化野生型多瘤病毒和猿猴病毒40颗粒的组蛋白H3和H4显示出广泛的乙酰化。源自多瘤病毒非转化宿主范围突变体的相同组蛋白组分未显示出这种高度的乙酰化。
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