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抗氧化剂对Atm基因缺陷小鼠癌症预防和神经运动性能的影响。

Effects of antioxidants on cancer prevention and neuromotor performance in Atm deficient mice.

作者信息

Reliene Ramune, Fleming Sheila M, Chesselet Marie-Françoise, Schiestl Robert H

机构信息

Department of Pathology, David Geffen School of Medicine and School of Public Health, UCLA, Los Angeles, CA 90095, USA.

出版信息

Food Chem Toxicol. 2008 Apr;46(4):1371-7. doi: 10.1016/j.fct.2007.08.028. Epub 2007 Aug 29.

Abstract

Ataxia telangiectasia (AT) is an autosomal recessive disorder characterized by immunodeficiency, neurodegeneration and cancer. The disease results from bi-allelic mutations in the AT mutated (ATM) gene involved in cell cycle checkpoint control and repair of DNA double-strand breaks. Evidence has been accumulating that oxidative stress is associated with AT and may be involved in the pathogenesis of the disease. This led to a hypothesis that antioxidants may alleviate the symptoms of AT. Consequently, several studies were conducted in Atm deficient mice to examine the role of antioxidants in cancer prevention and/or correction of neuromotor performance. N-acetyl-l-cysteine (NAC), EUK-189, tempol, and 5-carboxy-1,1,3,3-tetramethylisoindolin-2-yloxyl (CTMIO) have been tested in Atm deficient mice. In contrast to other antioxidants, NAC has been used in the clinical practice for many decades and is available as a dietary supplement. In this article, we review chemoprevention studies in Atm deficient mice and, in more detail, our findings on the effect of NAC. Our short-term study showed that NAC suppressed genome rearrangements linked to cancer. The long-term study demonstrated that NAC reduced the incidence and multiplicity of lymphoma and improved some aspects of motor performance.

摘要

共济失调毛细血管扩张症(AT)是一种常染色体隐性疾病,其特征为免疫缺陷、神经退行性变和癌症。该疾病由参与细胞周期检查点控制和DNA双链断裂修复的共济失调毛细血管扩张症突变(ATM)基因的双等位基因突变引起。越来越多的证据表明氧化应激与AT相关,并且可能参与该疾病的发病机制。这引发了一种假设,即抗氧化剂可能会减轻AT的症状。因此,人们在Atm基因缺陷小鼠中进行了多项研究,以检验抗氧化剂在癌症预防和/或神经运动性能纠正中的作用。N-乙酰-L-半胱氨酸(NAC)、EUK-189、tempol和5-羧基-1,1,3,3-四甲基异吲哚啉-2-氧基(CTMIO)已在Atm基因缺陷小鼠中进行了测试。与其他抗氧化剂不同,NAC已经在临床实践中使用了数十年,并且可以作为膳食补充剂获得。在本文中,我们回顾了在Atm基因缺陷小鼠中的化学预防研究,并更详细地阐述了我们关于NAC作用的研究结果。我们的短期研究表明,NAC抑制了与癌症相关的基因组重排。长期研究表明,NAC降低了淋巴瘤的发病率和多发性,并改善了运动性能的某些方面。

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