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本文引用的文献
1
A prevalent variant in PPP1R3A impairs glycogen synthesis and reduces muscle glycogen content in humans and mice.
PLoS Med. 2008 Jan 29;5(1):e27. doi: 10.1371/journal.pmed.0050027.
2
Cardiomyopathy and exercise intolerance in muscle glycogen storage disease 0.
N Engl J Med. 2007 Oct 11;357(15):1507-14. doi: 10.1056/NEJMoa066691.
3
Variation in GYS1 interacts with exercise and gender to predict cardiovascular mortality.
PLoS One. 2007 Mar 14;2(3):e285. doi: 10.1371/journal.pone.0000285.
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Glucose metabolism in mice lacking muscle glycogen synthase.
Diabetes. 2005 Dec;54(12):3466-73. doi: 10.2337/diabetes.54.12.3466.
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Digenic inheritance of severe insulin resistance in a human pedigree.
Nat Genet. 2002 Aug;31(4):379-84. doi: 10.1038/ng926. Epub 2002 Jul 15.
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The stimulation-induced increase in skeletal muscle glycogen synthase content is impaired in carriers of the glycogen synthase XbaI gene polymorphism.
Diabetes. 2001 Jan;50(1):195-8. doi: 10.2337/diabetes.50.1.195.
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A paired-sibling analysis of the XbaI polymorphism in the muscle glycogen synthase gene.
Diabetologia. 1999 Sep;42(9):1138-45. doi: 10.1007/s001250051282.
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Mutations in the liver glycogen synthase gene in children with hypoglycemia due to glycogen storage disease type 0.
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Association between polymorphism of the glycogen synthase gene and non-insulin-dependent diabetes mellitus.
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Early metabolic defects in persons at increased risk for non-insulin-dependent diabetes mellitus.
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