相似文献
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Reduced DNA repair in mouse satellite DNA after treatment with methylmethanesulfonate, and N-methyl-N-nitrosourea.
Nucleic Acids Res. 1976 Jul;3(7):1689-701. doi: 10.1093/nar/3.7.1689.
2
Carcinogen-induced DNA repair in nucleotide-permeable Escherichia coli cells. Induction of DNA repair by the carcinogens methyl and ethyl nitrosourea and methyl methanesulfonate.
Eur J Biochem. 1975 Aug 15;56(2):433-47. doi: 10.1111/j.1432-1033.1975.tb02250.x.
3
Nonuniform distribution of DNA repair in chromatin after treatment with methyl methanesulfonate.
Nucleic Acids Res. 1977 Aug;4(8):2619-28. doi: 10.1093/nar/4.8.2619.
4
Organ-specific effects of DNA methylation by alkylating agents in the inbred Swiss mouse.
Chem Biol Interact. 1975 Apr;10(4):285-93. doi: 10.1016/0009-2797(75)90093-9.
5
Postreplication repair of alkylation damage to DNA of mammalian cells in culture.
Cancer Res. 1975 Oct;35(10):2780-9.
6
Exhaustion and recovery of repair excision of O6-methylguanine from rat liver DNA.
Nature. 1976 Jan 15;259(5539):153-5. doi: 10.1038/259153a0.
7
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Effect of split doses of N-methyl-N-nitrosourea on DNA repair synthesis in cultured mammalian cells.
Cancer Lett. 1977 Sep;3(3-4):183-8. doi: 10.1016/s0304-3835(77)95492-1.
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[Increase in DNA repair after treatment of mice with N-methyl-N-nitrosourea].
Boll Soc Ital Biol Sper. 1980 Apr 30;56(8):745-50.
引用本文的文献
1
The use of radioimmunoassay to study the formation and disappearance of O6-methylguanine in mouse liver satellite and main-band DNA following dimethylnitrosamine administration.
J Cancer Res Clin Oncol. 1980;98(2):127-38. doi: 10.1007/BF00405957.
2
Potentiation of DNA adduct formation in HL-60 cells by combinations of benzene metabolites.
Proc Natl Acad Sci U S A. 1992 Aug 1;89(15):7105-9. doi: 10.1073/pnas.89.15.7105.
3
Distribution of 7-methylguanine and of replication sites in the different kinetic classes of DNA from rats treated with dimethylnitrosamine.
Nucleic Acids Res. 1978 Sep;5(9):3357-64. doi: 10.1093/nar/5.9.3357.
4
The influence of chromatin structure on the distribution of DNA repair synthesis studied by nuclease digestion.
Nucleic Acids Res. 1979 Jan;6(1):359-70. doi: 10.1093/nar/6.1.359.
5
Nonuniform distribution of DNA repair in chromatin after treatment with methyl methanesulfonate.
Nucleic Acids Res. 1977 Aug;4(8):2619-28. doi: 10.1093/nar/4.8.2619.
本文引用的文献
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Evidence for excision of ultraviolet-induced pyrimidine dimers from the DNA of human cells in vitro.
Biophys J. 1968 Mar;8(3):319-25. doi: 10.1016/S0006-3495(68)86490-2.
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Isolation and characterization of mouse and guinea pig satellite deoxyribonucleic acids.
Biochemistry. 1968 Dec;7(12):4373-9. doi: 10.1021/bi00852a033.
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Quantitative aspects of the repair of alkylated DNA in cultured mammalian cells. II. Non-semiconservative DNA synthesis ('repair synthesis') in HeLa and Chinese hamster cells following treatment with alkylating agents.
Chem Biol Interact. 1971 Feb;3(1):49-68. doi: 10.1016/0009-2797(71)90025-1.
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Studies on DNA repair in human lymphocytes treated with proximate carcinogens and alkylating agents.
Cancer Res. 1971 Sep;31(9):1297-306.
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Repeated sequences in DNA. Hundreds of thousands of copies of DNA sequences have been incorporated into the genomes of higher organisms.
Science. 1968 Aug 9;161(3841):529-40. doi: 10.1126/science.161.3841.529.
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Distribution of deoxyribonucleic acid repair synthesis among repetitive and unique sequences in the human diploid genome.
Biochemistry. 1974 Jul 16;13(15):3018-23. doi: 10.1021/bi00712a003.
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Mammary tumorigenesis in chemical carcinogen-treated mice. I. Incidence in BALB-c and C57BL mice.
J Natl Cancer Inst. 1974 Jul;53(1):213-21. doi: 10.1093/jnci/53.1.213.
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Preferential DNA repair in human cells.
Nature. 1974 Jan 4;247(5435):35-6. doi: 10.1038/247035a0.
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Fractionation of mouse DNA in preparative Ag+ -Cs2SO4 gradients.
Biochim Biophys Acta. 1973 Oct 26;324(3):309-19. doi: 10.1016/0005-2787(73)90277-3.
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Use of UV endonuclease from Micrococcus luteus to monitor the progress of DNA repair in UV-irradiated human cells.
Mutat Res. 1973 Aug;19(2):245-56. doi: 10.1016/0027-5107(73)90083-3.
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