• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

稻瘟病菌分离株对水稻和小麦宿主物种特异性的遗传分析。

Genetic Analysis of Host Species Specificity of Magnaporthe oryzae Isolates from Rice and Wheat.

出版信息

Phytopathology. 2006 May;96(5):480-4. doi: 10.1094/PHYTO-96-0480.

DOI:10.1094/PHYTO-96-0480
PMID:18944307
Abstract

ABSTRACT A Triticum isolate (pathogenic on wheat) of Magnaporthe oryzae was crossed with an Oryza isolate (pathogenic on rice) to elucidate mechanisms of their parasitic specificity on wheat. When the pathogenicity of their F (1) cultures (hybrids between a Triticum isolate and an Oryza isolate) was tested on wheat, avirulent and virulent cultures segregated in a 7:1 ratio. This result suggests that three loci are involved in avirulence of the Oryza isolate on wheat. One of the three loci conditioned papilla formation, whereas the others conditioned the hypersensitive reaction. Allelism tests revealed that the locus conditioning papilla formation is Pwt2 while one of the two loci conditioning the hypersensitive reaction is Pwt1. The other locus conditioning the hypersensitive reaction was different from any other known loci and, therefore, was designated as Pwt5.

摘要

摘要 本研究将稻瘟病菌(对水稻致病)的一个小麦分离株与一个水稻分离株进行杂交,以阐明其在小麦上寄生特异性的机制。当在小麦上测试它们的 F1(小麦分离株和水稻分离株之间的杂种)培养物的致病性时,无毒和有毒培养物以 7:1 的比例分离。这一结果表明,三个基因座参与了稻瘟病菌在小麦上的无毒。三个基因座中的一个控制着乳突的形成,而其他两个控制着过敏性反应。等位基因测试表明,控制乳突形成的基因座是 Pwt2,而控制过敏性反应的两个基因座中的一个是 Pwt1。控制过敏性反应的另一个基因座与任何其他已知基因座不同,因此被指定为 Pwt5。

相似文献

1
Genetic Analysis of Host Species Specificity of Magnaporthe oryzae Isolates from Rice and Wheat.稻瘟病菌分离株对水稻和小麦宿主物种特异性的遗传分析。
Phytopathology. 2006 May;96(5):480-4. doi: 10.1094/PHYTO-96-0480.
2
Analysis of Host Species Specificity of Magnaporthe grisea Toward Wheat Using a Genetic Cross Between Isolates from Wheat and Foxtail Millet.利用从小麦和谷子分离物间的遗传杂交分析灰霉菌对小麦的宿主种特异性。
Phytopathology. 2000 Oct;90(10):1060-7. doi: 10.1094/PHYTO.2000.90.10.1060.
3
PWT1, an avirulence gene of Magnaporthe oryzae tightly linked to the rDNA Locus, is recognized by two staple crops, common wheat and barley.PWT1 是稻瘟病菌的无毒基因,紧密连锁在 rDNA 基因座上,被两种主要作物,即普通小麦和大麦所识别。
Phytopathology. 2010 May;100(5):436-43. doi: 10.1094/PHYTO-100-5-0436.
4
Analysis of Host Species Specificity of Magnaporthe grisea Toward Foxtail Millet Using a Genetic Cross Between Isolates from Wheat and Foxtail Millet.利用从小麦和谷子分离菌系间的遗传杂交分析灰霉菌对谷子的种属特异性。
Phytopathology. 2003 Jan;93(1):42-5. doi: 10.1094/PHYTO.2003.93.1.42.
5
Identification of a novel locus Rmo2 conditioning resistance in barley to host-specific subgroups of Magnaporthe oryzae.鉴定大麦中一个新的 Rmo2 位点,该位点对稻瘟病菌特定宿主亚群具有抗性。
Phytopathology. 2012 Jul;102(7):674-82. doi: 10.1094/PHYTO-09-11-0256.
6
A Gene-for-Gene Relationship Underlying the Species-Specific Parasitism of Avena/Triticum Isolates of Magnaporthe grisea on Wheat Cultivars.灰霉菌 Avena/Triticum 分离株对小麦品种的种特异性寄生的基因对基因关系。
Phytopathology. 2002 Nov;92(11):1182-8. doi: 10.1094/PHYTO.2002.92.11.1182.
7
Genetic analysis and molecular mapping of the avirulence gene PRE1, a gene for host-species specificity in the blast fungus Magnaporthe grisea.稻瘟病菌中一个决定寄主物种特异性的无毒基因PRE1的遗传分析与分子定位
Genome. 2006 Aug;49(8):873-81. doi: 10.1139/g06-043.
8
Development of genomic SSR markers and molecular characterization of Magnaporthe oryzae isolates from wheat in Brazil.巴西小麦稻瘟病菌株的基因组SSR标记开发及分子特征分析
Biochem Genet. 2014 Feb;52(1-2):52-70. doi: 10.1007/s10528-013-9627-4. Epub 2013 Nov 23.
9
AVR1-CO39 is a predominant locus governing the broad avirulence of Magnaporthe oryzae 2539 on cultivated rice (Oryza sativa L.).AVR1-CO39 是控制稻瘟病菌 2539 对栽培稻广谱无毒的主要位点。
Mol Plant Microbe Interact. 2011 Jan;24(1):13-7. doi: 10.1094/MPMI-10-09-0240.
10
Genetic Analysis of Avirulence/Virulence of an Isolate of Magnaporthe grisea from a Rice Field in Texas.德克萨斯州稻田中稻瘟病菌分离物的无毒/毒性的遗传分析。
Phytopathology. 1997 Jan;87(1):71-6. doi: 10.1094/PHYTO.1997.87.1.71.

引用本文的文献

1
The ACE1 secondary metabolite gene cluster is a pathogenicity factor of wheat blast fungus.ACE1 次生代谢物基因簇是小麦赤霉病菌的致病因子。
Commun Biol. 2024 Jul 4;7(1):812. doi: 10.1038/s42003-024-06517-7.
2
Pyricularia oryzae: Lab star and field scourge.稻瘟病菌:实验室明星与田间祸害。
Mol Plant Pathol. 2024 Apr;25(4):e13449. doi: 10.1111/mpp.13449.
3
Wheat blast: A review from a genetic and genomic perspective.小麦稻瘟病:从遗传和基因组角度的综述
Front Microbiol. 2022 Sep 8;13:983243. doi: 10.3389/fmicb.2022.983243. eCollection 2022.
4
Pre and post stage of infection of Magnaporthe oryzae Oryza in wheat leaves with different resistance levels.稻瘟病菌在不同抗性水平小麦叶片上的侵染前和侵染后阶段。
Braz J Microbiol. 2022 Sep;53(3):1091-1100. doi: 10.1007/s42770-022-00749-7. Epub 2022 Apr 12.
5
Wheat Blast: A Disease Spreading by Intercontinental Jumps and Its Management Strategies.小麦条锈病:一种通过洲际传播扩散的病害及其管理策略。
Front Plant Sci. 2021 Jul 23;12:710707. doi: 10.3389/fpls.2021.710707. eCollection 2021.
6
The Impact of Blast Disease: Past, Present, and Future.爆炸病的影响:过去、现在和未来。
Methods Mol Biol. 2021;2356:1-18. doi: 10.1007/978-1-0716-1613-0_1.
7
Comparative Pathogenicity and Host Ranges of and Related Species.[具体物种名称]及相关物种的比较致病性和宿主范围
Plant Pathol J. 2020 Aug 1;36(4):305-313. doi: 10.5423/PPJ.FT.04.2020.0068.
8
Pyricularia graminis-tritici is not the correct species name for the wheat blast fungus: response to Ceresini et al. (MPP 20:2).小麦稻瘟病菌的正确物种名称不是Pyricularia graminis-tritici:对Ceresini等人(《植物病理学年评》20:2)的回应。
Mol Plant Pathol. 2019 Feb;20(2):173-179. doi: 10.1111/mpp.12778.
9
Comparative Methods for Molecular Determination of Host-Specificity Factors in Plant-Pathogenic Fungi.植物病原真菌寄主专化性因子的分子鉴定的比较方法。
Int J Mol Sci. 2018 Mar 15;19(3):863. doi: 10.3390/ijms19030863.
10
Gene Flow between Divergent Cereal- and Grass-Specific Lineages of the Rice Blast Fungus .稻瘟病菌中分歧的谷物和草特异性谱系间的基因流动。
mBio. 2018 Feb 27;9(1):e01219-17. doi: 10.1128/mBio.01219-17.