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A notch in the toll belt.

作者信息

Hertzog Paul

机构信息

Centre for Innate Immunity and Infectious Disease, Monash Institute of Medical Research, 27-31 Wright Street, Clayton, Victoria, Australia.

出版信息

Immunity. 2008 Nov 14;29(5):663-5. doi: 10.1016/j.immuni.2008.10.006.

DOI:10.1016/j.immuni.2008.10.006
PMID:19006687
Abstract

Two cell response pathways, Toll-like receptor (TLR) and Notch, conserved from Drosophila to mammals are well characterized for distinct roles in innate immunity and cell development, respectively. In this issue of Immunity, Hu et al. (2008) describe and characterize the direct cooperation of these two pathways.

摘要

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引用本文的文献

[1]
Insulin Modulates -Induced Inflammation by Restoring the Populations of NK Cells, Dendritic Cells, and B Lymphocytes in Lungs.

J Diabetes Res. 2018-10-22

[2]
The Critical Role of Notch1-TLR 4 Signaling in the Inflammatory and Fungicidal Activity of Macrophages Against Paracoccidioides brasiliensis Strain Pb18.

Mycopathologia. 2017-6-9

[3]
Notch and PKC are involved in formation of the lateral region of the dorso-ventral axis in Drosophila embryos.

PLoS One. 2013-7-4

[4]
Epigenetic control of cytomegalovirus latency and reactivation.

Viruses. 2013-5-23

[5]
Notch signal suppresses Toll-like receptor-triggered inflammatory responses in macrophages by inhibiting extracellular signal-regulated kinase 1/2-mediated nuclear factor κB activation.

J Biol Chem. 2011-12-28

[6]
Identification of novel targets of CSL-dependent Notch signaling in hematopoiesis.

PLoS One. 2011-5-26

[7]
Biphasic recruitment of transcriptional repressors to the murine cytomegalovirus major immediate-early promoter during the course of infection in vivo.

J Virol. 2010-1-27

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