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肉毒杆菌毒素相关蛋白的一种新功能:HA蛋白破坏肠道上皮屏障以增加毒素吸收。

A novel function of botulinum toxin-associated proteins: HA proteins disrupt intestinal epithelial barrier to increase toxin absorption.

作者信息

Fujinaga Y, Matsumura T, Jin Y, Takegahara Y, Sugawara Y

机构信息

Laboratory for Infection Cell Biology, International Research Center for Infectious Diseases, Research Institute for Microbial Diseases, Osaka University, Yamada-oka 3-1, Suita, Osaka 565-0871, Japan.

出版信息

Toxicon. 2009 Oct;54(5):583-6. doi: 10.1016/j.toxicon.2008.11.014. Epub 2008 Dec 6.

Abstract

Food-borne botulinum neurotoxin (BoNT) in the gastrointestinal lumen must cross an epithelial barrier to reach peripheral nerves to mediate its toxicity. The detailed mechanism by which BoNT traverses this barrier remains unclear. We found that hemagglutinin (HA) proteins of type B BoNT complex play an important role in the intestinal absorption of BoNT, disrupting the paracellular barrier of intestinal epithelium, which facilitates transepithelial delivery of BoNT both in vitro and in vivo (Matsumura, T., et al., 2008. Cell. Microbiol. 10, 355-364). We also found that type A HA proteins have a similar disrupting activity with a greater potency than type B HA proteins in the human intestinal epithelial cell lines Caco-2 and T84. In contrast, type C HA proteins in the toxin complex (up to 300 nM) have no detectable effect on the paracellular barrier in these human cell lines. These results may indicate that types A and B HA contribute to develop the food-borne human botulism by facilitating the intestinal transepithelial delivery of BoNTs.

摘要

胃肠道腔中的食源性肉毒杆菌神经毒素(BoNT)必须穿过上皮屏障才能到达外周神经以介导其毒性。BoNT穿过该屏障的详细机制仍不清楚。我们发现,B型BoNT复合物的血凝素(HA)蛋白在BoNT的肠道吸收中起重要作用,破坏肠上皮的细胞旁屏障,这在体外和体内均促进了BoNT的跨上皮递送(Matsumura,T.等人,2008年。细胞微生物学。10,355 - 364)。我们还发现,在人肠上皮细胞系Caco - 2和T84中,A型HA蛋白具有类似的破坏活性,且效力比B型HA蛋白更强。相比之下,毒素复合物中的C型HA蛋白(高达300 nM)对这些人细胞系中的细胞旁屏障没有可检测到 的影响。这些结果可能表明,A型和B型HA通过促进BoNTs的肠道跨上皮递送,有助于食源性人类肉毒中毒 的发生。

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