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Na+-mediated coupling between AMPA receptors and KNa channels shapes synaptic transmission.
Proc Natl Acad Sci U S A. 2008 Dec 30;105(52):20941-6. doi: 10.1073/pnas.0806403106. Epub 2008 Dec 18.
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Mechanisms of modulation of AMPA-induced Na+-activated K+ current by mGluR1.
J Neurophysiol. 2010 Jan;103(1):441-5. doi: 10.1152/jn.00584.2009. Epub 2009 Nov 4.
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Evidence for low GluR2 AMPA receptor subunit expression at synapses in the rat basolateral amygdala.
J Neurochem. 2005 Sep;94(6):1728-38. doi: 10.1111/j.1471-4159.2005.03334.x. Epub 2005 Jul 25.
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Activation of silent synapses by rapid activity-dependent synaptic recruitment of AMPA receptors.
J Neurosci. 2001 Aug 15;21(16):6008-17. doi: 10.1523/JNEUROSCI.21-16-06008.2001.
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Temporospatial coupling of networked synaptic activation of AMPA-type glutamate receptor channels and calcium transients in cultured motoneurons.
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Synaptic strengthening through activation of Ca2+-permeable AMPA receptors.
Nature. 1996 Jun 27;381(6585):793-6. doi: 10.1038/381793a0.

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2
A molecular switch in RCK2 triggers sodium-dependent activation of K1.1 (KCNT1) potassium channels.
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3
Disease-causing Slack potassium channel mutations produce opposite effects on excitability of excitatory and inhibitory neurons.
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The Na-activated K channel Slack contributes to synaptic development and plasticity.
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Structure-Based Identification and Characterization of Inhibitors of the Epilepsy-Associated K1.1 (KCNT1) Potassium Channel.
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An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human iPSC-Derived Neurons by Increasing Slack K Currents.
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1
Sodium-dependent potassium channels of a Slack-like subtype contribute to the slow afterhyperpolarization in lamprey spinal neurons.
J Physiol. 2007 Nov 15;585(Pt 1):75-90. doi: 10.1113/jphysiol.2007.138156. Epub 2007 Sep 20.
2
Characterization of Na+-activated K+ currents in larval lamprey spinal cord neurons.
J Neurophysiol. 2007 May;97(5):3484-93. doi: 10.1152/jn.00742.2006. Epub 2007 Feb 28.
4
High-conductance potassium channels of the SLO family.
Nat Rev Neurosci. 2006 Dec;7(12):921-31. doi: 10.1038/nrn1992.
5
Pharmacological activation and inhibition of Slack (Slo2.2) channels.
Neuropharmacology. 2006 Sep;51(4):896-906. doi: 10.1016/j.neuropharm.2006.06.003. Epub 2006 Jul 28.
6
For K+ channels, Na+ is the new Ca2+.
Trends Neurosci. 2005 Aug;28(8):422-8. doi: 10.1016/j.tins.2005.06.003.
7
SK channels in excitability, pacemaking and synaptic integration.
Curr Opin Neurobiol. 2005 Jun;15(3):305-11. doi: 10.1016/j.conb.2005.05.001.
8
Localization of the Na+-activated K+ channel Slick in the rat central nervous system.
J Comp Neurol. 2005 Mar 28;484(1):80-92. doi: 10.1002/cne.20462.
9
Slick (Slo2.1), a rapidly-gating sodium-activated potassium channel inhibited by ATP.
J Neurosci. 2003 Dec 17;23(37):11681-91. doi: 10.1523/JNEUROSCI.23-37-11681.2003.
10
Slo2 sodium-activated K+ channels bind to the PDZ domain of PSD-95.
Biochem Biophys Res Commun. 2003 Oct 31;310(4):1140-7. doi: 10.1016/j.bbrc.2003.09.133.

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