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Apoptotic functions of PDCD10/CCM3, the gene mutated in cerebral cavernous malformation 3.
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2
Differential angiogenesis function of CCM2 and CCM3 in cerebral cavernous malformations.
Neurosurg Focus. 2010 Sep;29(3):E1. doi: 10.3171/2010.5.FOCUS1090.
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Exceptional aggressiveness of cerebral cavernous malformation disease associated with PDCD10 mutations.
Genet Med. 2015 Mar;17(3):188-196. doi: 10.1038/gim.2014.97. Epub 2014 Aug 14.
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PDCD10 gene mutations in multiple cerebral cavernous malformations.
PLoS One. 2014 Oct 29;9(10):e110438. doi: 10.1371/journal.pone.0110438. eCollection 2014.
7
PDCD10, the gene mutated in cerebral cavernous malformation 3, is expressed in the neurovascular unit.
Neurosurgery. 2008 Apr;62(4):930-8; discussion 938. doi: 10.1227/01.neu.0000318179.02912.ca.
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Genomic causes of multiple cerebral cavernous malformations in a Japanese population.
J Clin Neurosci. 2013 May;20(5):667-9. doi: 10.1016/j.jocn.2012.05.041. Epub 2013 Feb 26.
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Mutations in apoptosis-related gene, PDCD10, cause cerebral cavernous malformation 3.
Neurosurgery. 2005 Nov;57(5):1008-13. doi: 10.1227/01.neu.0000180811.56157.e1.
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Endothelial cell clonal expansion in the development of cerebral cavernous malformations.
Nat Commun. 2019 Jun 24;10(1):2761. doi: 10.1038/s41467-019-10707-x.

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Cerebral Cavernous Malformation: From Genetics to Pharmacotherapy.
Brain Behav. 2025 Jan;15(1):e70223. doi: 10.1002/brb3.70223.
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The Dual Role of PDCD10 in Cancers: A Promising Therapeutic Target.
Cancers (Basel). 2022 Dec 3;14(23):5986. doi: 10.3390/cancers14235986.
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Inwardly rectifying potassium channels mediate polymyxin-induced nephrotoxicity.
Cell Mol Life Sci. 2022 May 15;79(6):296. doi: 10.1007/s00018-022-04316-z.
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The multifaceted gene.
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Genetic syndromes with vascular malformations - update on molecular background and diagnostics.
Arch Med Sci. 2020 Feb 25;17(4):965-991. doi: 10.5114/aoms.2020.93260. eCollection 2021.
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PDCD10-Deficiency Promotes Malignant Behaviors and Tumor Growth via Triggering EphB4 Kinase Activity in Glioblastoma.
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The Dysregulation and Prognostic Analysis of STRIPAK Complex Across Cancers.
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Antibodies in cerebral cavernous malformations react with cytoskeleton autoantigens in the lesional milieu.
J Autoimmun. 2020 Sep;113:102469. doi: 10.1016/j.jaut.2020.102469. Epub 2020 Apr 30.

本文引用的文献

1
PDCD10, the gene mutated in cerebral cavernous malformation 3, is expressed in the neurovascular unit.
Neurosurgery. 2008 Apr;62(4):930-8; discussion 938. doi: 10.1227/01.neu.0000318179.02912.ca.
2
ccm1 cell autonomously regulates endothelial cellular morphogenesis and vascular tubulogenesis in zebrafish.
Hum Mol Genet. 2008 Aug 15;17(16):2424-32. doi: 10.1093/hmg/ddn142. Epub 2008 May 10.
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KRIT-1/CCM1 is a Rap1 effector that regulates endothelial cell cell junctions.
J Cell Biol. 2007 Oct 22;179(2):247-54. doi: 10.1083/jcb.200705175.
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Krit 1 interactions with microtubules and membranes are regulated by Rap1 and integrin cytoplasmic domain associated protein-1.
FEBS J. 2007 Nov;274(21):5518-32. doi: 10.1111/j.1742-4658.2007.06068.x. Epub 2007 Oct 3.
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Proteomic identification of the cerebral cavernous malformation signaling complex.
J Proteome Res. 2007 Nov;6(11):4343-55. doi: 10.1021/pr0704276. Epub 2007 Sep 27.
7
Living with p53, dying of p53.
Cell. 2007 Aug 24;130(4):597-600. doi: 10.1016/j.cell.2007.08.005.
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CCM3 interacts with CCM2 indicating common pathogenesis for cerebral cavernous malformations.
Neurogenetics. 2007 Nov;8(4):249-56. doi: 10.1007/s10048-007-0098-9. Epub 2007 Jul 27.
9
PDCD10 interacts with Ste20-related kinase MST4 to promote cell growth and transformation via modulation of the ERK pathway.
Mol Biol Cell. 2007 Jun;18(6):1965-78. doi: 10.1091/mbc.e06-07-0608. Epub 2007 Mar 14.
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Genetics of cavernous angiomas.
Lancet Neurol. 2007 Mar;6(3):237-44. doi: 10.1016/S1474-4422(07)70053-4.

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