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MERIT40调控BRCA1-Rap80复合物的完整性并使其募集至DNA双链断裂处。

MERIT40 controls BRCA1-Rap80 complex integrity and recruitment to DNA double-strand breaks.

作者信息

Shao Genze, Patterson-Fortin Jeffrey, Messick Troy E, Feng Dan, Shanbhag Niraj, Wang Yingqun, Greenberg Roger A

机构信息

Department of Cancer Biology, Abramson Family Cancer Research Institute, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania 19104, USA.

出版信息

Genes Dev. 2009 Mar 15;23(6):740-54. doi: 10.1101/gad.1739609. Epub 2009 Mar 4.

Abstract

Rap80 targets the breast cancer suppressor protein BRCA1 along with Abraxas and the BRCC36 deubiquitinating enzyme (DUB) to polyubiquitin structures at DNA double-strand breaks (DSBs). These DSB targeting events are essential for BRCA1-dependent DNA damage response-induced checkpoint and repair functions. Here, we identify MERIT40 (Mediator of Rap80 Interactions and Targeting 40 kD)/(C19orf62) as a Rap80-associated protein that is essential for BRCA1-Rap80 complex protein interactions, stability, and DSB targeting. Moreover, MERIT40 is required for Rap80-associated lysine(63)-ubiquitin DUB activity, a critical component of BRCA1-Rap80 G2 checkpoint and viability responses to ionizing radiation. Thus, MERIT40 represents a novel factor that links BRCA1-Rap80 complex integrity, DSB recognition, and ubiquitin chain hydrolytic activities to the DNA damage response. These findings provide new molecular insights into how BRCA1 associates with independently assembled core protein complexes to maintain genome integrity.

摘要

Rap80与Abraxas及BRCC36去泛素化酶(DUB)一起,将乳腺癌抑制蛋白BRCA1靶向DNA双链断裂(DSB)处的多聚泛素结构。这些DSB靶向事件对于BRCA1依赖的DNA损伤反应诱导的检查点和修复功能至关重要。在此,我们鉴定出MERIT40(Rap80相互作用及靶向的40 kD介质)/(C19orf62)为一种与Rap80相关的蛋白质,它对于BRCA1-Rap80复合蛋白的相互作用、稳定性及DSB靶向至关重要。此外,MERIT40是Rap80相关的赖氨酸(63)-泛素DUB活性所必需的,而该活性是BRCA1-Rap80 G2检查点及对电离辐射的生存能力反应的关键组成部分。因此,MERIT40代表了一个将BRCA1-Rap80复合蛋白的完整性、DSB识别及泛素链水解活性与DNA损伤反应联系起来的新因子。这些发现为BRCA1如何与独立组装的核心蛋白复合物结合以维持基因组完整性提供了新的分子见解。

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