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视网膜脉络膜(视睫状)分流静脉、失明与视神经萎缩:慢性视神经受压的非特异性体征

Retinochoroidal (optociliary) shunt veins, blindness and optic atrophy: a non-specific sign of chronic optic nerve compression.

作者信息

Miller N R, Solomon S

机构信息

Neuro-Ophthalmology Unit, Johns Hopkins Hospital, Baltimore, Maryland.

出版信息

Aust N Z J Ophthalmol. 1991 May;19(2):105-9. doi: 10.1111/j.1442-9071.1991.tb00636.x.

DOI:10.1111/j.1442-9071.1991.tb00636.x
PMID:1930992
Abstract

Fifteen patients are described in whom the triad of blindness, optic disc swelling followed by optic atrophy, and optociliary shunt veins occurred. The causes of the syndrome included spheno-orbital meningioma, optic nerve glioma, meningocele of the optic nerve, and chronic papilloedema. It is postulated that chronic compression of the intraorbital portion of the optic nerve produces gradual obstruction of the central retinal vein, thus preventing the normal passage of venous blood from the retina through the central retinal vein to the cavernous sinus. Optociliary veins are a pre-existing shunt system that allows retinal venous blood to bypass the central retinal vein and exit from the orbit via the choroidal circulation and its anastomoses.

摘要

本文描述了15例出现失明、视盘肿胀继以视神经萎缩以及视神经睫状静脉三联征的患者。该综合征的病因包括蝶眶脑膜瘤、视神经胶质瘤、视神经脑膜膨出和慢性视乳头水肿。据推测,视神经眶内段的慢性压迫导致视网膜中央静脉逐渐阻塞,从而阻止静脉血从视网膜正常通过视网膜中央静脉流入海绵窦。视神经睫状静脉是一个预先存在的分流系统,它允许视网膜静脉血绕过视网膜中央静脉,通过脉络膜循环及其吻合支从眼眶流出。

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Retinochoroidal (optociliary) shunt veins, blindness and optic atrophy: a non-specific sign of chronic optic nerve compression.视网膜脉络膜(视睫状)分流静脉、失明与视神经萎缩:慢性视神经受压的非特异性体征
Aust N Z J Ophthalmol. 1991 May;19(2):105-9. doi: 10.1111/j.1442-9071.1991.tb00636.x.
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Optociliary veins, disc pallor and visual loss. A triad of signs indicating spheno-orbital meningioma.眼睫状静脉、视盘苍白和视力丧失。提示蝶眶脑膜瘤的三联征。
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Optociliary shunt vessels in compressive optic neuropathy by the intracranial internal carotid artery.颅内颈内动脉所致压迫性视神经病变中的光睫状分流血管
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引用本文的文献

1
Disappearance of optociliary shunt vessels after optic nerve sheath decompression.视神经鞘减压术后视睫状分流血管消失
Br J Ophthalmol. 1996 Feb;80(2):186-7. doi: 10.1136/bjo.80.2.186-a.