Neville R G, Greenblatt S H, Kollartis C R
J Clin Neuroophthalmol. 1984 Jun;4(2):97-101.
We describe a 51-year-old woman with a falx meningioma causing Foster Kennedy syndrome accompanied by optociliary shunt vessels on the atrophic optic nerve. The tumor did not directly involve optic nerves or chiasm. The optociliary vessels disappeared after successful tumor removal. The patient's arteriograms, surgical findings, and postoperative course support the theory that optic atrophy in Foster Kennedy syndrome may be caused by compression of the optic nerve by adjacent brain tissue displaced by a distant intracranial mass lesion.
我们描述了一名51岁女性,患有镰旁脑膜瘤,导致福斯特·肯尼迪综合征,并伴有萎缩性视神经上的睫状分流血管。肿瘤未直接累及视神经或视交叉。成功切除肿瘤后,睫状血管消失。患者的动脉造影、手术结果及术后病程支持以下理论:福斯特·肯尼迪综合征中的视神经萎缩可能是由远处颅内占位性病变移位的相邻脑组织对视神经的压迫所致。