Abdrakhmanov A A, Aĭtkhozhina G K
Radiobiologiia. 1990 Sep-Oct;30(5):647-50.
With radiation injury caused by superlethal doses of 150 Gy the analgesic effect of narcotic drugs decreases and the level of drug analgesia depends on the initial status of nociceptive reactions. The role of mu-opiate mechanisms in the development of postirradiation analgesia to thermal stimuli has been determined.
在150戈瑞超致死剂量引起的辐射损伤情况下，麻醉药物的镇痛效果会降低，且药物镇痛水平取决于伤害性反应的初始状态。已确定μ-阿片机制在辐射后对热刺激镇痛作用发展中的作用。
