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本文引用的文献

1
Detection of mixed clarithromycin-resistant and -susceptible Helicobacter pylori using nested PCR and direct sequencing of DNA extracted from faeces.使用巢式PCR和对粪便中提取的DNA进行直接测序来检测克拉霉素耐药和敏感的幽门螺杆菌混合菌株
J Med Microbiol. 2007 Sep;56(Pt 9):1174-1180. doi: 10.1099/jmm.0.47302-0.
2
Efficacy of clarithromycin-based triple therapy for treating Helicobacter pylori in Thai non-ulcer dyspeptic patients with clarithromycin-resistant strains.基于克拉霉素的三联疗法治疗泰国克拉霉素耐药菌株非溃疡性消化不良患者幽门螺杆菌的疗效
J Med Assoc Thai. 2006 Sep;89 Suppl 3:S74-8.
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Mutations in the 23S rRNA gene of clarithromycin-resistant Helicobacter pylori from Japan.来自日本的克拉霉素耐药幽门螺杆菌23S rRNA基因中的突变
Int J Antimicrob Agents. 2007 Sep;30(3):250-4. doi: 10.1016/j.ijantimicag.2007.04.009. Epub 2007 Jun 21.
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Causal role of Helicobacter pylori infection in gastric cancer: an Asian enigma.幽门螺杆菌感染在胃癌中的因果作用：一个亚洲谜团。
World J Gastroenterol. 2006 Mar 7;12(9):1346-51. doi: 10.3748/wjg.v12.i9.1346.
5
Simple method for determination of the number of Helicobacter pylori CagA variable-region EPIYA tyrosine phosphorylation motifs by PCR.通过聚合酶链反应（PCR）测定幽门螺杆菌CagA可变区EPIYA酪氨酸磷酸化基序数量的简单方法。
J Clin Microbiol. 2005 Feb;43(2):791-5. doi: 10.1128/JCM.43.2.791-795.2005.
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Tyrosine phosphorylation of CagA from Chinese Helicobacter pylori isolates in AGS gastric epithelial cells.中国幽门螺杆菌分离株的CagA在AGS胃上皮细胞中的酪氨酸磷酸化
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Association between diversity in the Src homology 2 domain--containing tyrosine phosphatase binding site of Helicobacter pylori CagA protein and gastric atrophy and cancer.幽门螺杆菌CagA蛋白Src同源2结构域结合酪氨酸磷酸酶位点的多样性与胃萎缩及癌症之间的关联
J Infect Dis. 2004 Mar 1;189(5):820-7. doi: 10.1086/381782. Epub 2004 Feb 2.
8
Biological activity of the Helicobacter pylori virulence factor CagA is determined by variation in the tyrosine phosphorylation sites.幽门螺杆菌毒力因子CagA的生物活性由酪氨酸磷酸化位点的变异决定。
Proc Natl Acad Sci U S A. 2002 Oct 29;99(22):14428-33. doi: 10.1073/pnas.222375399. Epub 2002 Oct 21.
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Helicobacter pylori and gastrointestinal tract adenocarcinomas.幽门螺杆菌与胃肠道腺癌
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10
Cloning and sequence analysis of two copies of a 23S rRNA gene from Helicobacter pylori and association of clarithromycin resistance with 23S rRNA mutations.幽门螺杆菌23S rRNA基因两个拷贝的克隆与序列分析以及克拉霉素耐药性与23S rRNA突变的关联
Antimicrob Agents Chemother. 1997 Dec;41(12):2621-8. doi: 10.1128/AAC.41.12.2621.

利用泰国儿童粪便分析幽门螺杆菌克拉霉素耐药性和 CagA 状态。

Analysis of clarithromycin resistance and CagA status in Helicobacter pylori by use of feces from children in Thailand.

机构信息

Department of Microbiology, School of Pharmacy, Tokyo University of Pharmacy and Life Sciences, Hachioji, Tokyo, Japan.

出版信息

J Clin Microbiol. 2009 Dec;47(12):4144-5. doi: 10.1128/JCM.00786-09. Epub 2009 Sep 30.

DOI:10.1128/JCM.00786-09
PMID:19794037
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2786625/
Abstract

The clarithromycin resistance and CagA status of Helicobacter pylori in Thai children were investigated using fecal samples. Of the 284 samples, H. pylori was detected in 120 samples, and the clarithromycin resistance rate was 29.2%. The cagA gene was detected in 59 samples, and only 6.8% of these samples contained the East Asian CagA type.

摘要

本研究采用粪便样本检测泰国儿童幽门螺杆菌的克拉霉素耐药性和 CagA 状态。在 284 份样本中，120 份样本检测到幽门螺杆菌，克拉霉素耐药率为 29.2%。59 份样本检测到 cagA 基因，其中仅 6.8%的样本含有东亚 CagA 型。