镉离子对金黄色葡萄球菌中[14C]谷氨酸转运的能量供体依赖性效应。
Energy donor-dependent effect of Cd2+ on [14C]glutamate transport in Staphylococcus aureus.
作者信息
Malm A, Tynecka Z
机构信息
Department of Pharmaceutical Microbiology, Medical Academy, Lublin, Poland.
出版信息
Acta Biochim Pol. 1990;37(1):117-20.
In starved cells of Cd2(+)-sensitive Staphylococcus aureus 17810S preloaded with either glutamate or pyruvate, [14C]glutamate transport was blocked by 10 microM Cd2+, whereas in cells preloaded with lactate, [14C]glutamate transport was not affected. This differential effect of Cd2+ could be due to the presence or absence of dithiols in the substrate oxidizing systems. In starved cells of Cd2(+)-resistant strain 17810R preloaded with either of the three substrates, [14C]glutamate transport was insensitive to 10 microM Cd2+.
在预先加载了谷氨酸或丙酮酸的对镉离子敏感的金黄色葡萄球菌17810S的饥饿细胞中，10微摩尔的镉离子会阻断[14C]谷氨酸的转运，而在预先加载了乳酸的细胞中，[14C]谷氨酸的转运不受影响。镉离子的这种差异效应可能是由于底物氧化系统中存在或不存在二硫醇。在预先加载了三种底物之一的对镉离子有抗性的菌株17810R的饥饿细胞中，[14C]谷氨酸的转运对10微摩尔的镉离子不敏感。
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