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创伤愈合中形成的纤维组织的起源和结构。

THE ORIGIN AND STRUCTURE OF A FIBROUS TISSUE FORMED IN WOUND HEALING.

机构信息

Osborn Zoological Laboratory of Yale University, New Haven.

出版信息

J Exp Med. 1916 Jun 1;23(6):739-56. doi: 10.1084/jem.23.6.739.

DOI:10.1084/jem.23.6.739
PMID:19868020
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2125451/
Abstract
  1. In experimental wounds, made by removing various sized pieces of skin from the frog, there is a rapid coagulation of the blood plasma and lymph to form a coagulation tissue which fills the wound cavity. 2. The observations on the living animals show that the coagulation a grave intoxication, but a low reading may be observed in some fatal cases and gives no assurance that a fatal intoxication may not supervene. The kidneys in practically all these experiments are normal in all respects. It is possible that protein or tissue destruction rather than impaired eliminative function is responsible for the rise in non-coagulable nitrogen of the blood in these acute intoxications. Transfusions of dextrose solutions often benefit intestinal obstruction, and may depress the level of the non-coagulable nitrogen in the blood. Some cases show no change in non-coagulable nitrogen following transfusions and diuresis, and, as a rule, such cases present the most severe intoxication.
摘要
  1. 在通过从青蛙身上切除不同大小的皮肤块而制成的实验性伤口中,血浆和淋巴迅速凝结形成凝结组织,填充伤口腔。

  2. 对活体动物的观察表明,凝结是严重中毒的标志,但在某些致命病例中可能观察到低读数,并不能保证不会发生致命中毒。实际上,在所有这些实验中,肾脏在各方面均正常。在这些急性中毒中,血液中非凝结性氮的升高可能是由于蛋白质或组织破坏而不是排泄功能受损所致。葡萄糖溶液的输血通常有益于肠梗阻,并可能降低血液中非凝结性氮的水平。有些病例在输血和利尿后非凝结性氮没有变化,而且通常情况下,这种病例的中毒最严重。

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J Exp Med. 1921 Nov 30;34(6):625-42. doi: 10.1084/jem.34.6.625.

本文引用的文献

1
THE ORIGIN AND STRUCTURE OF A FIBROUS TISSUE WHICH APPEARS IN LIVING CULTURES OF ADULT FROG TISSUES.活体外层成年蛙组织中出现的纤维状组织的起源和结构。
J Exp Med. 1915 May 1;21(5):455-78. doi: 10.1084/jem.21.5.455.