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Elucidating factors underlying parent-offspring similarity in eating pathology in pre- and early puberty: Exploring the possibility of passive gene-environment correlation.探讨青少年前期和早期进食障碍中亲代-子代相似性的潜在因素:探索被动基因-环境关联的可能性。
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本文引用的文献

1
Molecular Genetic Studies of Eating Disorders: Current Status and Future Directions.饮食失调的分子遗传学研究:现状与未来方向
Curr Dir Psychol Sci. 2007 Feb 1;16(1):37-41. doi: 10.1111/j.1467-8721.2007.00471.x.
2
Puberty and the genetic diathesis of disordered eating attitudes and behaviors.青春期与饮食障碍态度和行为的遗传素质。
J Abnorm Psychol. 2009 Nov;118(4):788-96. doi: 10.1037/a0017207.
3
Rethinking environmental contributions to child and adolescent psychopathology: a meta-analysis of shared environmental influences.重新审视环境对儿童和青少年精神病理学的影响:共享环境影响的元分析
Psychol Bull. 2009 Jul;135(4):608-37. doi: 10.1037/a0015702.
4
Do weight and shape concerns exhibit genetic effects? Investigating discrepant findings.体重和体型担忧是否存在遗传效应?探究不一致的发现。
Int J Eat Disord. 2010 Jan;43(1):29-34. doi: 10.1002/eat.20666.
5
An investigation of temperament endophenotype candidates for early emergence of the core cognitive component of eating disorders.对饮食失调核心认知成分早期出现的气质内表型候选因素的调查。
Psychol Med. 2009 May;39(5):811-21. doi: 10.1017/S0033291708004261. Epub 2008 Aug 28.
6
The eating disorder examination: norms and construct validity with young and middle adolescent girls.饮食失调检查:针对青少年中、晚期女孩的常模与结构效度
Int J Eat Disord. 2008 Sep;41(6):551-8. doi: 10.1002/eat.20526.
7
Prenatal hormone exposure and risk for eating disorders: a comparison of opposite-sex and same-sex twins.产前激素暴露与饮食失调风险:异性双胞胎与同性双胞胎的比较
Arch Gen Psychiatry. 2008 Mar;65(3):329-36. doi: 10.1001/archgenpsychiatry.2007.47.
8
Ovarian hormones and binge eating: exploring associations in community samples.卵巢激素与暴饮暴食:探索社区样本中的关联
Psychol Med. 2008 Dec;38(12):1749-57. doi: 10.1017/S0033291708002997. Epub 2008 Feb 29.
9
Familiality and heritability of binge eating disorder: results of a case-control family study and a twin study.暴饮暴食症的家族性与遗传性:一项病例对照家庭研究和一项双生子研究的结果
Int J Eat Disord. 2008 Mar;41(2):174-9. doi: 10.1002/eat.20484.
10
Changes in genetic and environmental influences on disordered eating across adolescence: a longitudinal twin study.青少年期饮食失调的遗传和环境影响变化:一项纵向双胞胎研究。
Arch Gen Psychiatry. 2007 Dec;64(12):1409-15. doi: 10.1001/archpsyc.64.12.1409.

年龄对体重和体型担忧的遗传和环境影响的差异。

Age differences in genetic and environmental influences on weight and shape concerns.

机构信息

Department of Psychology, Michigan State University, East Lansing, Michigan 48824-1116, USA.

出版信息

Int J Eat Disord. 2010 Dec;43(8):679-88. doi: 10.1002/eat.20772.

DOI:10.1002/eat.20772
PMID:19950189
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2891330/
Abstract

OBJECTIVE

Previous research has shown important developmental shifts ingenetic and environmental influences for disordered eating. However, little research has examined age differences for weight/shape concerns, two key components of eating disorders. The goal of this study was to investigate these age differences in preadolescent, adolescent, young adult, and mid-adult twins.

METHOD

Participants included 2,618 female twins (ages of 10-41 years) from three large twin registries. Shape and weight concerns were assessed with the Eating Disorders Examination Questionnaire.

RESULTS

Genetic influences were modest in preadolescent twins, but significant from early-adolescence through middle adulthood. Shared environmental factors showed the opposite pattern, with the largest shared environmental contributions occurring in the youngest age group. Nonshared environmental effects remained relatively constant across age.

DISCUSSION

Findings highlight the importance of age differences in genetic and environmental influences. Possible mechanisms include gene x environment interactions and biological changes associated with key developmental stages.

摘要

目的

先前的研究表明,在饮食失调方面,遗传和环境影响存在重要的发展转变。然而,很少有研究探讨体重/体型担忧的年龄差异,这是饮食失调的两个关键组成部分。本研究的目的是调查青少年前、青少年、年轻成人和中年成年人双胞胎之间的这些年龄差异。

方法

参与者包括来自三个大型双胞胎登记处的 2618 名女性双胞胎(年龄在 10 至 41 岁之间)。使用饮食失调检查问卷评估体型和体重担忧。

结果

遗传影响在青少年前双胞胎中较小,但从青少年早期到中年成人期则显著。共同环境因素则呈现相反的模式,最大的共同环境贡献发生在最年轻的年龄组。非共享环境效应在整个年龄范围内相对稳定。

讨论

研究结果强调了遗传和环境影响在年龄方面的差异的重要性。可能的机制包括基因 x 环境相互作用和与关键发育阶段相关的生物学变化。