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空气污染与加那利群岛的死亡率:一项时间序列分析。

Air pollution and mortality in the Canary Islands: a time-series analysis.

机构信息

Dirección General de Salud Pública, Gobierno de Canarias, Alfonso XIII, Las Palmas de Gran Canaria, Spain.

出版信息

Environ Health. 2010 Feb 12;9:8. doi: 10.1186/1476-069X-9-8.

DOI:10.1186/1476-069X-9-8
PMID:20152037
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2843667/
Abstract

BACKGROUND

The island factor of the cities of Las Palmas de Gran Canaria and Santa Cruz de Tenerife, along with their proximity to Africa and their meteorology, create a particular setting that influences the air quality of these cities and provides researchers an opportunity to analyze the acute effects of air-pollutants on daily mortality.

METHODS

From 2000 to 2004, the relationship between daily changes in PM10, PM2.5, SO2, NO2, CO, and ozone levels and daily total mortality and mortality due to respiratory and heart diseases were assessed using Generalized Additive Poisson models controlled for potential confounders. The lag effect (up to five days) as well as the concurrent and previous day averages and distributed lag models were all estimated. Single and two pollutant models were also constructed.

RESULTS

Daily levels of PM10, PM2.5, NO2, and SO2 were found to be associated with an increase in respiratory mortality in Santa Cruz de Tenerife and with increased heart disease mortality in Las Palmas de Gran Canaria, thus indicating an association between daily ozone levels and mortality from heart diseases. The effects spread over five successive days. SO2 was the only air pollutant significantly related with total mortality (lag 0).

CONCLUSIONS

There is a short-term association between current exposure levels to air pollution and mortality (total as well as that due specifically to heart and respiratory diseases) in both cities. Risk coefficients were higher for respiratory and cardiovascular mortality, showing a delayed effect over several days.

摘要

背景

拉斯帕尔马斯德大加那利岛和圣克鲁斯德特内里费市的岛屿因素,加上它们与非洲的接近程度和气象条件,形成了一个特殊的环境,影响着这些城市的空气质量,为研究人员分析空气污染物对每日死亡率的急性影响提供了机会。

方法

2000 年至 2004 年,使用广义加性泊松模型控制潜在混杂因素,评估了 PM10、PM2.5、SO2、NO2、CO 和臭氧水平的日变化与总死亡率以及呼吸系统和心脏病死亡率之间的关系。估计了滞后效应(长达五天)以及同期和前一天的平均值和分布滞后模型。还构建了单污染物和双污染物模型。

结果

发现 PM10、PM2.5、NO2 和 SO2 的日水平与圣克鲁斯德特内里费市呼吸系统死亡率的增加以及拉斯帕尔马斯德大加那利岛心脏病死亡率的增加有关,这表明每日臭氧水平与心脏病死亡率之间存在关联。影响持续五天。SO2 是唯一与总死亡率(lag 0)显著相关的空气污染物。

结论

在这两个城市,当前暴露于空气污染水平与死亡率(包括心脏病和呼吸系统疾病的死亡率)之间存在短期关联。呼吸和心血管死亡率的风险系数更高,显示出几天的滞后效应。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/b90375e37db6/1476-069X-9-8-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/3e3ada6b5cc7/1476-069X-9-8-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/c9c20fde368a/1476-069X-9-8-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/b90375e37db6/1476-069X-9-8-3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/3e3ada6b5cc7/1476-069X-9-8-1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/c9c20fde368a/1476-069X-9-8-2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b000/2843667/b90375e37db6/1476-069X-9-8-3.jpg

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本文引用的文献

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