免疫球蛋白与疱疹病毒诱导的Fc受体结合可抑制病毒生长。 - Suppr

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超能文献

Immunoglobulin binding to herpes virus-induced Fc receptors inhibits virus growth.

作者信息

Costa J, Rabson A S, Yee C, Tralka T S

出版信息

Nature. 1977 Sep 15;269(5625):251-2. doi: 10.1038/269251a0.

DOI:10.1038/269251a0

PMID:201851

Abstract

摘要

相似文献

Immunoglobulin binding to herpes virus-induced Fc receptors inhibits virus growth.免疫球蛋白与疱疹病毒诱导的Fc受体结合可抑制病毒生长。

Nature. 1977 Sep 15;269(5625):251-2. doi: 10.1038/269251a0.

Role of Fc receptors in herpes simplex virus infection.

Lancet. 1975 Jan 11;1(7898):77-8. doi: 10.1016/s0140-6736(75)91077-6.

Characteristics of the Fc receptor induced by herpes simplex virus.单纯疱疹病毒诱导的Fc受体的特征

Intervirology. 1978;10(1):32-9. doi: 10.1159/000148965.

Characterization of herpes simplex virus type 1-induced Fc receptor in its interaction with rabbit immunoglobulin G (IgG).1型单纯疱疹病毒诱导的Fc受体与兔免疫球蛋白G（IgG）相互作用的特性研究

APMIS. 1990 Aug;98(8):685-94. doi: 10.1111/j.1699-0463.1990.tb04989.x.

The mononuclear cell in human blood which mediates antibody-dependent cellular cytotoxicity to virus-infected target cells. II. Identification as a K cell.人血液中的单核细胞介导对病毒感染靶细胞的抗体依赖性细胞毒性作用。II. 鉴定为K细胞。

J Immunol. 1977 Feb;118(2):567-73.

Further immunological evidence for the presence of Fc receptors on the surface of HSV-infected cells.单纯疱疹病毒（HSV）感染细胞表面存在Fc受体的进一步免疫学证据。

Virologie. 1976 Oct-Dec;27(4):239-43.

Immunoglobulin G(Fc)-binding receptors on virions of herpes simplex virus type 1 and transfer of these receptors to the cell surface by infection.1型单纯疱疹病毒病毒粒子上的免疫球蛋白G（Fc）结合受体以及通过感染将这些受体转移至细胞表面

J Virol. 1980 May;34(2):512-20. doi: 10.1128/JVI.34.2.512-520.1980.

Characterization of regions of herpes simplex virus type 1 glycoprotein E involved in binding the Fc domain of monomeric IgG and in forming a complex with glycoprotein I.1型单纯疱疹病毒糖蛋白E中参与结合单体IgG的Fc结构域以及与糖蛋白I形成复合物的区域的特征分析

J Immunol. 1995 Jan 1;154(1):260-7.

Interaction between herpes simplex type 1-induced Fc receptor and human and rabbit immunoglobulin G (IgG) domains.单纯疱疹病毒1型诱导的Fc受体与人及兔免疫球蛋白G（IgG）结构域之间的相互作用。

Immunology. 1986 Jun;58(2):251-5.

Absence of host-cell influence on binding specificity of herpes simplex virus type 1 induced Fc receptor.

Acta Pathol Microbiol Immunol Scand C. 1987 Jun;95(3):113-6. doi: 10.1111/j.1699-0463.1987.tb00017.x.

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Complement depletion facilitates the infection of multiple brain tumors by an intravascular, replication-conditional herpes simplex virus mutant.补体耗竭促进血管内复制条件性单纯疱疹病毒突变体对多种脑肿瘤的感染。

J Virol. 2000 May;74(10):4765-75. doi: 10.1128/jvi.74.10.4765-4775.2000.

In vivo immune evasion mediated by the herpes simplex virus type 1 immunoglobulin G Fc receptor.由1型单纯疱疹病毒免疫球蛋白G Fc受体介导的体内免疫逃逸

J Virol. 1998 Jul;72(7):5351-9. doi: 10.1128/JVI.72.7.5351-5359.1998.

Response to X-radiation and cytotoxic drugs of clonal subpopulations of different ploidy and metastatic potential isolated from RIF-1 mouse sarcoma.从RIF-1小鼠肉瘤中分离出的不同倍性和转移潜能的克隆亚群对X射线和细胞毒性药物的反应。

Br J Cancer. 1983 Jun;47(6):841-8. doi: 10.1038/bjc.1983.139.

Human immunoglobulin class and subclass specificity of Fc receptors induced by herpes simplex virus type 1.1型单纯疱疹病毒诱导的Fc受体的人免疫球蛋白类别和亚类特异性

J Virol. 1984 Jun;50(3):796-804. doi: 10.1128/JVI.50.3.796-804.1984.

Inhibition of herpes simplex virus replication by succinyl concanavalin A.琥珀酰伴刀豆球蛋白A对单纯疱疹病毒复制的抑制作用

Antimicrob Agents Chemother. 1982 Mar;21(3):450-5. doi: 10.1128/AAC.21.3.450.

Effect of immunosuppression on recurrent herpes simplex in mice.免疫抑制对小鼠复发性单纯疱疹的影响。

Infect Immun. 1980 Sep;29(3):902-7. doi: 10.1128/iai.29.3.902-907.1980.

Antibody-mediated destruction of virus-infected cells.抗体介导的病毒感染细胞的破坏。

Adv Immunol. 1980;29:209-60. doi: 10.1016/s0065-2776(08)60045-0.

J Virol. 1980 May;34(2):512-20. doi: 10.1128/JVI.34.2.512-520.1980.

Differences in antigenic properties of Fc-binding activity during infection with herpes simplex virus type 1.单纯疱疹病毒1型感染期间Fc结合活性抗原特性的差异。

Arch Virol. 1984;80(2-3):183-93. doi: 10.1007/BF01310658.

Monoclonal antibodies suppress replication of herpes simplex virus type 1 in trigeminal ganglia.单克隆抗体抑制单纯疱疹病毒1型在三叉神经节中的复制。

J Virol. 1984 Sep;51(3):656-61. doi: 10.1128/JVI.51.3.656-661.1984.

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