相似文献
1
Energy crosstalk between DNA lesions: implications for allosteric coupling of DNA repair and triplet repeat expansion pathways.
J Am Chem Soc. 2010 Mar 31;132(12):4095-7. doi: 10.1021/ja1002857.
2
Energetic coupling between clustered lesions modulated by intervening triplet repeat bulge loops: allosteric implications for DNA repair and triplet repeat expansion.
Biopolymers. 2010 Apr;93(4):355-69. doi: 10.1002/bip.21343.
3
Dynamic DNA Energy Landscapes and Substrate Complexity in Triplet Repeat Expansion and DNA Repair.
Biomolecules. 2019 Nov 6;9(11):709. doi: 10.3390/biom9110709.
4
DNA repair and DNA triplet repeat expansion: the impact of abasic lesions on triplet repeat DNA energetics.
J Am Chem Soc. 2009 Jul 8;131(26):9354-60. doi: 10.1021/ja902161e.
5
Structure and dynamics in DNA looped domains: CAG triplet repeat sequence dynamics probed by 2-aminopurine fluorescence.
Biochemistry. 2007 Sep 25;46(38):10756-66. doi: 10.1021/bi7005674. Epub 2007 Aug 25.
6
Energy landscapes of dynamic ensembles of rolling triplet repeat bulge loops: implications for DNA expansion associated with disease states.
J Am Chem Soc. 2012 Apr 4;134(13):6033-44. doi: 10.1021/ja3010896. Epub 2012 Mar 23.
7
Conformational energetics of stable and metastable states formed by DNA triplet repeat oligonucleotides: implications for triplet expansion diseases.
Proc Natl Acad Sci U S A. 2002 Nov 12;99(23):14700-5. doi: 10.1073/pnas.222519799. Epub 2002 Nov 4.
8
Impact of bulge loop size on DNA triplet repeat domains: Implications for DNA repair and expansion.
Biopolymers. 2014 Jan;101(1):1-12. doi: 10.1002/bip.22236.
9
Trinucleotide repeat DNA alters structure to minimize the thermodynamic impact of 8-oxo-7,8-dihydroguanine.
Biochemistry. 2012 Jan 10;51(1):52-62. doi: 10.1021/bi201552s. Epub 2011 Dec 14.
10
The impact of an exocyclic cytosine adduct on DNA duplex properties: significant thermodynamic consequences despite modest lesion-induced structural alterations.
Biochemistry. 1998 Sep 8;37(36):12507-12. doi: 10.1021/bi981090b.
引用本文的文献
1
Political science meets physical science: The shared concept of stability.
PNAS Nexus. 2023 Dec 12;2(12):pgad401. doi: 10.1093/pnasnexus/pgad401. eCollection 2023 Dec.
2
Dynamic DNA Energy Landscapes and Substrate Complexity in Triplet Repeat Expansion and DNA Repair.
Biomolecules. 2019 Nov 6;9(11):709. doi: 10.3390/biom9110709.
3
In What Ways Do Synthetic Nucleotides and Natural Base Lesions Alter the Structural Stability of G-Quadruplex Nucleic Acids?
J Nucleic Acids. 2017;2017:1641845. doi: 10.1155/2017/1641845. Epub 2017 Oct 18.
4
Loss of loop adenines alters human telomere d[AG3(TTAG3)3] quadruplex folding.
Nucleic Acids Res. 2014 Dec 16;42(22):14031-41. doi: 10.1093/nar/gku1245. Epub 2014 Nov 26.
5
APE1 incision activity at abasic sites in tandem repeat sequences.
J Mol Biol. 2014 May 29;426(11):2183-98. doi: 10.1016/j.jmb.2014.03.014. Epub 2014 Apr 1.
6
Impact of bulge loop size on DNA triplet repeat domains: Implications for DNA repair and expansion.
Biopolymers. 2014 Jan;101(1):1-12. doi: 10.1002/bip.22236.
7
Energy landscapes of dynamic ensembles of rolling triplet repeat bulge loops: implications for DNA expansion associated with disease states.
J Am Chem Soc. 2012 Apr 4;134(13):6033-44. doi: 10.1021/ja3010896. Epub 2012 Mar 23.
8
Trinucleotide repeat DNA alters structure to minimize the thermodynamic impact of 8-oxo-7,8-dihydroguanine.
Biochemistry. 2012 Jan 10;51(1):52-62. doi: 10.1021/bi201552s. Epub 2011 Dec 14.
9
On the sequence-directed nature of human gene mutation: the role of genomic architecture and the local DNA sequence environment in mediating gene mutations underlying human inherited disease.
Hum Mutat. 2011 Oct;32(10):1075-99. doi: 10.1002/humu.21557. Epub 2011 Sep 2.
本文引用的文献
1
Characterization of a K+-induced conformational switch in a human telomeric DNA oligonucleotide using 2-aminopurine fluorescence.
Biochemistry. 2010 Jan 12;49(1):179-94. doi: 10.1021/bi901357r.
2
Thermodynamic aspects of coupled binding and folding of an intrinsically disordered protein: a computational alanine scanning study.
Biochemistry. 2009 Dec 8;48(48):11332-4. doi: 10.1021/bi901705z.
3
Energetic coupling between clustered lesions modulated by intervening triplet repeat bulge loops: allosteric implications for DNA repair and triplet repeat expansion.
Biopolymers. 2010 Apr;93(4):355-69. doi: 10.1002/bip.21343.
4
The origin of allosteric functional modulation: multiple pre-existing pathways.
Structure. 2009 Aug 12;17(8):1042-50. doi: 10.1016/j.str.2009.06.008.
5
DNA repair and DNA triplet repeat expansion: the impact of abasic lesions on triplet repeat DNA energetics.
J Am Chem Soc. 2009 Jul 8;131(26):9354-60. doi: 10.1021/ja902161e.
6
Non-B DNA conformations as determinants of mutagenesis and human disease.
Mol Carcinog. 2009 Apr;48(4):273-85. doi: 10.1002/mc.20507.
7
Protein allostery, signal transmission and dynamics: a classification scheme of allosteric mechanisms.
Mol Biosyst. 2009 Mar;5(3):207-16. doi: 10.1039/b819720b. Epub 2009 Jan 6.
8
The yield, processing, and biological consequences of clustered DNA damage induced by ionizing radiation.
J Radiat Res. 2009 Jan;50(1):27-36. doi: 10.1269/jrr.08086.
9
DNA energy landscapes via calorimetric detection of microstate ensembles of metastable macrostates and triplet repeat diseases.
Proc Natl Acad Sci U S A. 2008 Nov 25;105(47):18326-30. doi: 10.1073/pnas.0810376105. Epub 2008 Nov 17.
10
Allostery and cooperativity revisited.
Protein Sci. 2008 Aug;17(8):1295-307. doi: 10.1110/ps.03259908. Epub 2008 Jun 17.
Zotero 插件