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Positioning NK-kappaB in multiple myeloma.多发性骨髓瘤中核因子κB的定位
Blood. 2010 Apr 29;115(17):3422-4. doi: 10.1182/blood-2010-01-264796.
2
Targeting of CXCR3 improves anti-myeloma efficacy of adoptively transferred activated natural killer cells.靶向 CXCR3 可提高过继转移激活自然杀伤细胞治疗骨髓瘤的疗效。
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Fratricide of NK Cells in Daratumumab Therapy for Multiple Myeloma Overcome by -Expanded Autologous NK Cells.达雷妥尤单抗治疗多发性骨髓瘤导致 NK 细胞亲缘性杀伤被扩增的自体 NK 细胞所克服。
Clin Cancer Res. 2018 Aug 15;24(16):4006-4017. doi: 10.1158/1078-0432.CCR-17-3117. Epub 2018 Apr 17.
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Multiple Myeloma Impairs Bone Marrow Localization of Effector Natural Killer Cells by Altering the Chemokine Microenvironment.多发性骨髓瘤通过改变趋化因子微环境来损害效应自然杀伤细胞在骨髓中的定位。
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GSK-3β/β-catenin pathway plays crucial roles in the regulation of NK cell cytotoxicity against myeloma cells.GSK-3β/β-catenin 通路在调节 NK 细胞对骨髓瘤细胞的细胞毒性方面发挥着关键作用。
FASEB J. 2023 Mar;37(3):e22821. doi: 10.1096/fj.202201658RR.
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Harnessing natural killer cells for the treatment of multiple myeloma.利用自然杀伤细胞治疗多发性骨髓瘤。
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Natural killer cell lines preferentially kill clonogenic multiple myeloma cells and decrease myeloma engraftment in a bioluminescent xenograft mouse model.自然杀伤细胞系优先杀伤集落形成性多发性骨髓瘤细胞,并降低生物发光异种移植小鼠模型中的骨髓瘤嵌合体。
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The proteasome inhibitor bortezomib disrupts tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) expression and natural killer (NK) cell killing of TRAIL receptor-positive multiple myeloma cells.蛋白酶体抑制剂硼替佐米破坏肿瘤坏死因子相关凋亡诱导配体(TRAIL)的表达和自然杀伤(NK)细胞对 TRAIL 受体阳性多发性骨髓瘤细胞的杀伤作用。
Mol Immunol. 2010 Aug;47(14):2388-96. doi: 10.1016/j.molimm.2010.05.003. Epub 2010 Jun 9.
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Natural killer cells efficiently target multiple myeloma clonogenic tumor cells.自然杀伤细胞能有效地靶向多发性骨髓瘤集落形成肿瘤细胞。
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Long-chain polyunsaturated omega-3 fatty acids reduce multiple myeloma exosome-mediated suppression of NK cell cytotoxicity.长链多不饱和ω-3 脂肪酸可减少多发性骨髓瘤外泌体介导的 NK 细胞细胞毒性抑制作用。
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Constraints on signaling network logic reveal functional subgraphs on Multiple Myeloma OMIC data.信号网络逻辑的限制揭示了多发性骨髓瘤组学数据上的功能子图。
BMC Syst Biol. 2018 Mar 21;12(Suppl 3):32. doi: 10.1186/s12918-018-0551-4.
2
Logic programming reveals alteration of key transcription factors in multiple myeloma.逻辑编程揭示多发性骨髓瘤中关键转录因子的改变。
Sci Rep. 2017 Aug 23;7(1):9257. doi: 10.1038/s41598-017-09378-9.
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Aspirin and risk of multiple myeloma in adults: A systematic review and meta-analysis.阿司匹林与成人多发性骨髓瘤风险:一项系统评价与荟萃分析
Leuk Res Rep. 2017 Feb 28;7:23-28. doi: 10.1016/j.lrr.2017.02.002. eCollection 2017.
4
Toll-like receptor (TLR)-1/2 triggering of multiple myeloma cells modulates their adhesion to bone marrow stromal cells and enhances bortezomib-induced apoptosis.Toll样受体(TLR)-1/2激活多发性骨髓瘤细胞可调节其与骨髓基质细胞的黏附,并增强硼替佐米诱导的细胞凋亡。
PLoS One. 2014 May 2;9(5):e96608. doi: 10.1371/journal.pone.0096608. eCollection 2014.
5
Serum deprivation elevates the levels of microvesicles with different size distributions and selectively enriched proteins in human myeloma cells in vitro.血清剥夺可提高体外培养的人骨髓瘤细胞中具有不同大小分布且蛋白质选择性富集的微泡水平。
Acta Pharmacol Sin. 2014 Mar;35(3):381-93. doi: 10.1038/aps.2013.166. Epub 2013 Dec 30.
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Drug resistance in multiple myeloma: latest findings and new concepts on molecular mechanisms.多发性骨髓瘤中的耐药性:分子机制的最新发现与新概念
Oncotarget. 2013 Dec;4(12):2186-207. doi: 10.18632/oncotarget.1497.
7
Regular aspirin use and risk of multiple myeloma: a prospective analysis in the health professionals follow-up study and nurses' health study.常规使用阿司匹林与多发性骨髓瘤风险:在卫生专业人员随访研究和护士健康研究中的前瞻性分析。
Cancer Prev Res (Phila). 2014 Jan;7(1):33-41. doi: 10.1158/1940-6207.CAPR-13-0224. Epub 2013 Nov 26.
8
Disruption of IkappaB kinase (IKK)-mediated RelA serine 536 phosphorylation sensitizes human multiple myeloma cells to histone deacetylase (HDAC) inhibitors.IKK 介导的 RelA 丝氨酸 536 磷酸化的破坏使人类多发性骨髓瘤细胞对组蛋白去乙酰化酶(HDAC)抑制剂敏感。
J Biol Chem. 2011 Sep 30;286(39):34036-50. doi: 10.1074/jbc.M111.284216. Epub 2011 Aug 4.

本文引用的文献

1
Classical and/or alternative NF-kappaB pathway activation in multiple myeloma.多发性骨髓瘤中经典和/或替代 NF-κB 通路的激活。
Blood. 2010 Apr 29;115(17):3541-52. doi: 10.1182/blood-2009-09-243535. Epub 2010 Jan 6.
2
Atacicept in relapsed/refractory multiple myeloma or active Waldenström's macroglobulinemia: a phase I study.阿他西普用于复发/难治性多发性骨髓瘤或活动性华氏巨球蛋白血症:一项I期研究。
Br J Cancer. 2009 Oct 6;101(7):1051-8. doi: 10.1038/sj.bjc.6605241.
3
Regulation and function of NF-kappaB transcription factors in the immune system.NF-κB转录因子在免疫系统中的调控与功能
Annu Rev Immunol. 2009;27:693-733. doi: 10.1146/annurev.immunol.021908.132641.
4
The role of IGF-1 as a major growth factor for myeloma cell lines and the prognostic relevance of the expression of its receptor.胰岛素样生长因子-1作为骨髓瘤细胞系主要生长因子的作用及其受体表达的预后相关性。
Blood. 2009 May 7;113(19):4614-26. doi: 10.1182/blood-2008-07-170464. Epub 2009 Feb 18.
5
Plasma cell differentiation and survival.浆细胞分化与存活
Curr Opin Immunol. 2008 Apr;20(2):162-9. doi: 10.1016/j.coi.2008.03.016. Epub 2008 May 2.
6
Promiscuous mutations activate the noncanonical NF-kappaB pathway in multiple myeloma.杂乱突变激活多发性骨髓瘤中的非经典核因子κB通路。
Cancer Cell. 2007 Aug;12(2):131-44. doi: 10.1016/j.ccr.2007.07.003.
7
Frequent engagement of the classical and alternative NF-kappaB pathways by diverse genetic abnormalities in multiple myeloma.多发性骨髓瘤中多种遗传异常频繁激活经典和替代NF-κB信号通路。
Cancer Cell. 2007 Aug;12(2):115-30. doi: 10.1016/j.ccr.2007.07.004.
8
Targeting NF-kappaB pathway with an IKK2 inhibitor induces inhibition of multiple myeloma cell growth.用IKK2抑制剂靶向NF-κB通路可诱导多发性骨髓瘤细胞生长受到抑制。
Br J Haematol. 2007 Jul;138(2):160-8. doi: 10.1111/j.1365-2141.2007.06629.x. Epub 2007 Jun 3.
9
BAFF and APRIL protect myeloma cells from apoptosis induced by interleukin 6 deprivation and dexamethasone.B细胞活化因子(BAFF)和增殖诱导配体(APRIL)可保护骨髓瘤细胞免受因白细胞介素6缺乏和地塞米松诱导的细胞凋亡。
Blood. 2004 Apr 15;103(8):3148-57. doi: 10.1182/blood-2003-06-1984. Epub 2003 Dec 4.
10
BCMA is essential for the survival of long-lived bone marrow plasma cells.BCMA对长寿骨髓浆细胞的存活至关重要。
J Exp Med. 2004 Jan 5;199(1):91-8. doi: 10.1084/jem.20031330.

Positioning NK-kappaB in multiple myeloma.

作者信息

Klein Bernard

机构信息

INSERM, u847, Montpellier, France.

出版信息

Blood. 2010 Apr 29;115(17):3422-4. doi: 10.1182/blood-2010-01-264796.

DOI:10.1182/blood-2010-01-264796
PMID:20430962
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2997801/
Abstract
摘要