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IL-17A has an important role in the acute attacks of Behçet's disease.

作者信息

Ekinci Nurten Sayin, Alpsoy Erkan, Karakas Ayse Akman, Yilmaz Senem Buyukkara, Yegin Olcay

出版信息

J Invest Dermatol. 2010 Aug;130(8):2136-8. doi: 10.1038/jid.2010.114. Epub 2010 May 6.

DOI:10.1038/jid.2010.114
PMID:20445554
Abstract
摘要

相似文献

1
IL-17A has an important role in the acute attacks of Behçet's disease.白细胞介素-17A在白塞病急性发作中起重要作用。
J Invest Dermatol. 2010 Aug;130(8):2136-8. doi: 10.1038/jid.2010.114. Epub 2010 May 6.
2
[Analysis of the profile of CD4+ cells in Behçet's disease].
Nippon Ganka Gakkai Zasshi. 1997 Apr;101(4):335-40.
3
Secretion of interleukin-17 by CD8+ T cells expressing CD146 (MCAM).CD8+T 细胞表达 CD146（MCAM）分泌白细胞介素-17。
Clin Immunol. 2014 May-Jun;152(1-2):36-47. doi: 10.1016/j.clim.2014.01.009. Epub 2014 Feb 13.
4
Increased frequencies of interleukin-2- and interferon-gamma-producing T cells in patients with active Behçet's disease.活动性白塞病患者中产生白细胞介素-2和干扰素-γ的T细胞频率增加。
Invest Ophthalmol Vis Sci. 1998 May;39(6):996-1004.
5
Interleukin-2, interleukin-6 and T regulatory cells in peripheral blood of patients with Behçet's disease and recurrent aphthous ulcerations.白塞病和复发性口腔溃疡患者外周血中白细胞介素 2、白细胞介素 6 和 T 调节细胞。
J Oral Pathol Med. 2012 Jan;41(1):73-9. doi: 10.1111/j.1600-0714.2011.01061.x. Epub 2011 Jul 8.
6
Increased entry of CD4+ T cells into the Th1 cytokine effector pathway during T-cell division following stimulation in Behcet's disease.白塞病中，刺激后T细胞分裂期间CD4+ T细胞进入Th1细胞因子效应途径的比例增加。
Rheumatology (Oxford). 2004 Jul;43(7):843-51. doi: 10.1093/rheumatology/keh195. Epub 2004 May 18.
7
[Expression and function of ICOS on CD4 T cells and application to therapy in patients with ocular Behçet's disease with uveitis].
Nippon Ganka Gakkai Zasshi. 2012 Nov;116(11):1037-45.
8
The number of CD8+ T cells and NKT cells increases in the aqueous humor of patients with Behçet's uveitis.白塞氏葡萄膜炎患者房水中CD8 + T细胞和自然杀伤T细胞数量增加。
Clin Exp Immunol. 2004 Aug;137(2):437-43. doi: 10.1111/j.1365-2249.2004.02536.x.
9
[Immunological study of Behcet's disease].
Bull Soc Pathol Exot Filiales. 1978 Mar-Apr;71(2):221-6.
10
Expression and function of inducible costimulator on peripheral blood CD4+ T cells in Behçet's patients with uveitis: a new activity marker?诱导性共刺激分子在白塞氏葡萄膜炎患者外周血CD4+ T细胞中的表达及功能：一种新的活动标志物？
Invest Ophthalmol Vis Sci. 2010 Oct;51(10):5099-104. doi: 10.1167/iovs.10-5286. Epub 2010 May 5.

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Associations of IL-17 and IL-17 receptor polymorphisms with Behçet's disease in Denizli Province of Turkey.土耳其代尼兹利省白细胞介素 17 和白细胞介素 17 受体多态性与白塞病的关联。
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2
Transcriptome analysis of immune cells from Behçet's syndrome patients: the importance of IL-17-producing cells and antigen-presenting cells in the pathogenesis of Behçet's syndrome.Behçet 综合征患者免疫细胞的转录组分析：IL-17 产生细胞和抗原提呈细胞在 Behçet 综合征发病机制中的重要性。
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Behçet Disease: An Update for Dermatologists.
白塞病：皮肤科医生的最新知识。
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Immune-Mediated Retinal Vasculitis in Posterior Uveitis and Experimental Models: The Leukotriene (LT)B4-VEGF Axis.后部葡萄膜炎中的免疫介导性视网膜血管炎和实验模型：白三烯 (LT)B4-VEGF 轴。
Cells. 2021 Feb 15;10(2):396. doi: 10.3390/cells10020396.
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Experimental Therapeutic Solutions for Behcet's Disease.白塞病的实验性治疗方案
J Exp Pharmacol. 2021 Feb 12;13:127-145. doi: 10.2147/JEP.S265645. eCollection 2021.
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[Progress in interferon: A treatment of Behcet syndrome].[干扰素的进展：白塞综合征的一种治疗方法]
Beijing Da Xue Xue Bao Yi Xue Ban. 2020 Dec 18;52(6):1166-1170. doi: 10.19723/j.issn.1671-167X.2020.06.032.
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Behçet's Disease: An Overview of Etiopathogenesis.白塞病：发病机制概述。
Front Immunol. 2019 May 10;10:1067. doi: 10.3389/fimmu.2019.01067. eCollection 2019.
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Incomplete intestinal obstruction as the possible main complaint in Behcet's disease after surgery for recurrent abdominal aortic pseudoaneurysms: a case report and literature review.术后复发性腹主动脉假性动脉瘤的 Behcet 病中以不完全性肠梗阻为主要表现：1 例病例报告并文献复习。
BMC Cardiovasc Disord. 2018 Dec 17;18(1):238. doi: 10.1186/s12872-018-0977-z.
9
The Role of Th17 Cells in the Pathogenesis of Behcet's Disease.辅助性T细胞17在白塞病发病机制中的作用
J Clin Med. 2017 Jul 21;6(7):74. doi: 10.3390/jcm6070074.
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Neuro-Behçet disease mimicking brain tumor: A case report.酷似脑肿瘤的神经白塞病：一例报告。
Surg Neurol Int. 2017 Jun 5;8:97. doi: 10.4103/sni.sni_3_17. eCollection 2017.