Research on early-stage carcinogenesis: are we approaching paradigm instability?
作者信息
Baker Stuart G, Cappuccio Antonio, Potter John D
机构信息
Biometry Research Group, Division of Cancer Prevention, National Cancer Institute, Bethesda, MD, USA.
出版信息
J Clin Oncol. 2010 Jul 10;28(20):3215-8. doi: 10.1200/JCO.2010.28.5460. Epub 2010 Jun 14.
Abstract
摘要
相似文献
1
Research on early-stage carcinogenesis: are we approaching paradigm instability?早期致癌作用的研究：我们是否正接近范式不稳定？
J Clin Oncol. 2010 Jul 10;28(20):3215-8. doi: 10.1200/JCO.2010.28.5460. Epub 2010 Jun 14.
2
Transient appearance of the mutator phenotype during carcinogenesis as a possible explanation for the lack of mini/microsatellite instability in many late stage tumors.在致癌过程中突变体表型的短暂出现，可能是许多晚期肿瘤缺乏微卫星/小卫星不稳定性的一种解释。
Mutat Res. 1998 Nov 3;421(2):221-5. doi: 10.1016/s0027-5107(98)00209-7.
3
A nonidentifiability aspect of the two-stage model of carcinogenesis.致癌作用两阶段模型的一个不可识别方面。
Risk Anal. 1996 Oct;16(5):711-5. doi: 10.1111/j.1539-6924.1996.tb00819.x.
4
Cancer studies clash over mechanisms of malignancy.癌症研究在恶性肿瘤发生机制上存在分歧。
Nature. 2015 Dec 17;528(7582):317. doi: 10.1038/528317a.
5
Alternating electric fields and carcinogenesis: a new paradigm to avoid missing the elephant in the room.交变电场与致癌作用：避免忽视关键问题的新范式。
World Neurosurg. 2015 May;83(5):718-22. doi: 10.1016/j.wneu.2015.03.003. Epub 2015 Mar 11.
6
The somatic mutation theory of cancer: growing problems with the paradigm?癌症的体细胞突变理论：该范式面临的问题日益增多？
Bioessays. 2004 Oct;26(10):1097-107. doi: 10.1002/bies.20087.
7
Theories of carcinogenesis: an emerging perspective.致癌理论：一种新出现的观点。
Semin Cancer Biol. 2008 Oct;18(5):372-7. doi: 10.1016/j.semcancer.2008.03.012. Epub 2008 Mar 26.
8
Gene mutation might contribute to carcinogenesis by disrupting the asymmetric division of somatic stem cells.基因突变可能通过破坏体细胞干细胞的不对称分裂而导致癌症发生。
Med Hypotheses. 2008;70(6):1230-1. doi: 10.1016/j.mehy.2008.01.026. Epub 2008 Mar 17.
9
Are times a' changin' in carcinogenesis?致癌作用中的时代正在改变吗？
Endocrinology. 2005 Jan;146(1):11-2. doi: 10.1210/en.2004-1376.
10
Mutation and cancer: a model for human carcinogenesis.突变与癌症：人类致癌作用的一种模型
J Natl Cancer Inst. 1981 Jun;66(6):1037-52. doi: 10.1093/jnci/66.6.1037.
引用本文的文献
1
Rethinking carcinogenesis: The detached pericyte hypothesis.重新思考癌症发生：脱离的周细胞假说。
Med Hypotheses. 2020 Nov;144:110056. doi: 10.1016/j.mehy.2020.110056. Epub 2020 Jun 30.
2
Systemic Metabolism, Its Regulators, and Cancer: Past Mistakes and Future Potential.全身代谢、其调节因子与癌症：过去的误区与未来的潜力
Front Endocrinol (Lausanne). 2019 Feb 12;10:65. doi: 10.3389/fendo.2019.00065. eCollection 2019.
3
The Significance of an Enhanced Concept of the Organism for Medicine.强化的机体概念对医学的意义。
Evid Based Complement Alternat Med. 2016;2016:1587652. doi: 10.1155/2016/1587652. Epub 2016 Jun 29.
4
Bioelectrical Signals and Ion Channels in the Modeling of Multicellular Patterns and Cancer Biophysics.多细胞模式建模与癌症生物物理学中的生物电信号和离子通道
Sci Rep. 2016 Feb 4;6:20403. doi: 10.1038/srep20403.
5
Cancer etiology. Variation in cancer risk among tissues can be explained by the number of stem cell divisions.癌症病因。组织间癌症风险的差异可由干细胞分裂次数来解释。
Science. 2015 Jan 2;347(6217):78-81. doi: 10.1126/science.1260825.
6
A cancer theory kerfuffle can lead to new lines of research.一场癌症理论的争论可能会引发新的研究方向。
J Natl Cancer Inst. 2014 Dec 20;107(2). doi: 10.1093/jnci/dju405. Print 2015 Feb.
7
Global consensus theorem and self-organized criticality: unifying principles for understanding self-organization, swarm intelligence and mechanisms of carcinogenesis.全球共识定理与自组织临界性：理解自组织、群体智能及致癌机制的统一原则。
Gene Regul Syst Bio. 2013;7:23-39. doi: 10.4137/GRSB.S10885. Epub 2013 Feb 20.
8
Human adipose tissue from normal and tumoral breast regulates the behavior of mammary epithelial cells.人正常和肿瘤乳腺组织调节乳腺上皮细胞的行为。
Clin Transl Oncol. 2013 Feb;15(2):124-31. doi: 10.1007/s12094-012-0896-x. Epub 2012 Jul 19.
9
Transcriptional output in a prospective design conditionally on follow-up and exposure: the multistage model of cancer.以前瞻性设计为条件，基于随访和暴露情况的转录输出：癌症的多阶段模型。
Int J Mol Epidemiol Genet. 2012;3(2):107-14. Epub 2012 May 10.
10
Morphogenetic fields in embryogenesis, regeneration, and cancer: non-local control of complex patterning.胚胎发生、再生和癌症中的形态发生场：复杂模式的非局部控制
Biosystems. 2012 Sep;109(3):243-61. doi: 10.1016/j.biosystems.2012.04.005. Epub 2012 Apr 20.
本文引用的文献
1
Chromosomes in leukemia and beyond: from irrelevant to central players.白血病及其他病症中的染色体：从无关因素到核心角色
Annu Rev Genomics Hum Genet. 2009;10:1-18. doi: 10.1146/annurev-genom-082908-150144.
2
The cancer genome.癌症基因组
Nature. 2009 Apr 9;458(7239):719-24. doi: 10.1038/nature07943.
3
Plausibility of stromal initiation of epithelial cancers without a mutation in the epithelium: a computer simulation of morphostats.上皮无突变情况下基质引发上皮癌的合理性：形态稳定态的计算机模拟
BMC Cancer. 2009 Mar 23;9:89. doi: 10.1186/1471-2407-9-89.
4
Tissue architecture and function: dynamic reciprocity via extra- and intra-cellular matrices.组织结构与功能：通过细胞外和细胞内基质实现的动态相互作用。
Cancer Metastasis Rev. 2009 Jun;28(1-2):167-76. doi: 10.1007/s10555-008-9178-z.
5
Advances in chemical carcinogenesis: a historical review and prospective.化学致癌作用的进展：历史回顾与展望
Cancer Res. 2008 Sep 1;68(17):6863-72. doi: 10.1158/0008-5472.CAN-08-2852.
6
Theories of carcinogenesis: an emerging perspective.致癌理论：一种新出现的观点。
Semin Cancer Biol. 2008 Oct;18(5):372-7. doi: 10.1016/j.semcancer.2008.03.012. Epub 2008 Mar 26.
7
Epigenetics in cancer.癌症中的表观遗传学
N Engl J Med. 2008 Mar 13;358(11):1148-59. doi: 10.1056/NEJMra072067.
8
Oncogenes and cancer.癌基因与癌症
N Engl J Med. 2008 Jan 31;358(5):502-11. doi: 10.1056/NEJMra072367.
9
Paradoxes in carcinogenesis: new opportunities for research directions.癌症发生中的悖论：研究方向的新机遇
BMC Cancer. 2007 Aug 6;7:151. doi: 10.1186/1471-2407-7-151.
10
Morphogens, morphostats, microarchitecture and malignancy.形态发生素、形态稳定素、微结构与恶性肿瘤
Nat Rev Cancer. 2007 Jun;7(6):464-74. doi: 10.1038/nrc2146.
Zotero 插件