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Hibernating myocardium: a mitochondrial adaptation that may be destined to heart failure.

作者信息

Thomas Sabu, McFalls Edward O

机构信息

Cardiology Section, VA Medical Center, University of Minnesota, Minneapolis, MN 55417, USA.

出版信息

J Cardiovasc Transl Res. 2008 Dec;1(4):328-31. doi: 10.1007/s12265-008-9069-2. Epub 2008 Oct 17.

DOI:10.1007/s12265-008-9069-2
PMID:20559945
Abstract
摘要

相似文献

1
Hibernating myocardium: a mitochondrial adaptation that may be destined to heart failure.冬眠心肌：一种可能注定导致心力衰竭的线粒体适应性变化。
J Cardiovasc Transl Res. 2008 Dec;1(4):328-31. doi: 10.1007/s12265-008-9069-2. Epub 2008 Oct 17.
2
Adaptation of nonrevascularized human hibernating and chronically stunned myocardium to long-term chronic myocardial ischemia.未行血运重建的人类冬眠及长期顿抑心肌对长期慢性心肌缺血的适应性
Am J Cardiol. 2006 Dec 15;98(12):1574-80. doi: 10.1016/j.amjcard.2006.07.035. Epub 2006 Oct 18.
3
[Hibernation and stunning as manifestations of ischemic dysfunction of the myocardium].[冬眠与心肌顿抑：心肌缺血性功能障碍的表现]
Kardiologiia. 2005;45(9):73-8.
4
[The basis of chronic ischemic reversible left ventricular dysfunction (hibernating myocardium)].[慢性缺血性可逆性左心室功能障碍（冬眠心肌）的基础]
Cesk Fysiol. 1999 Feb;48(1):4-8.
5
[Stunning, hibernation, and heart failure in patients with coronary disease: crucial role of impaired coronary flow reserve].
Ital Heart J Suppl. 2003 Sep;4(9):727-32.
6
Metabolic adaptation during a sequence of no-flow and low-flow ischemia. A possible trigger for hibernation.无血流和低血流缺血序列中的代谢适应。冬眠的一个可能触发因素。
Circulation. 1996 Nov 15;94(10):2587-96. doi: 10.1161/01.cir.94.10.2587.
7
From coronary artery disease to heart failure: role of the hibernating myocardium.
Am J Cardiol. 1995 Apr 27;75(13):16E-22E. doi: 10.1016/s0002-9149(99)80443-3.
8
[Hibernating and stunned myocardium. Pathogenetic mechanisms during and after myocardial ischemia].
Recenti Prog Med. 2002 Oct;93(10):565-8.
9
Ubiquity of myocardial stunning.心肌顿抑的普遍性。
Basic Res Cardiol. 1995 Jul-Aug;90(4):253-6. doi: 10.1007/BF00797892.
10
Hibernating myocardium: is the program to survive a pathway to failure?冬眠心肌：存活程序会成为走向衰竭的途径吗？
Circ Res. 2008 Jan 4;102(1):3-5. doi: 10.1161/CIRCRESAHA.107.168278.

本文引用的文献

1
Deficiency or inhibition of oxygen sensor Phd1 induces hypoxia tolerance by reprogramming basal metabolism.氧传感器Phd1的缺陷或抑制通过重新编程基础代谢诱导缺氧耐受性。
Nat Genet. 2008 Feb;40(2):170-80. doi: 10.1038/ng.2007.62. Epub 2008 Jan 6.
2
Ischemic preconditioning modulates mitochondrial respiration, irrespective of the employed signal transduction pathway.缺血预处理可调节线粒体呼吸，而与所采用的信号转导途径无关。
Transl Res. 2008 Jan;151(1):17-26. doi: 10.1016/j.trsl.2007.09.007. Epub 2007 Oct 29.
3
Persistent regional downregulation in mitochondrial enzymes and upregulation of stress proteins in swine with chronic hibernating myocardium.
慢性冬眠心肌猪线粒体酶的持续性局部下调及应激蛋白的上调。
Circ Res. 2008 Jan 4;102(1):103-12. doi: 10.1161/CIRCRESAHA.107.155895. Epub 2007 Oct 25.
4
The energetic state within hibernating myocardium is normal during dobutamine despite inhibition of ATP-dependent potassium channel opening with glibenclamide.在使用多巴酚丁胺期间，尽管用格列本脲抑制了ATP依赖性钾通道开放，但冬眠心肌内的能量状态正常。
Am J Physiol Heart Circ Physiol. 2007 Nov;293(5):H2945-51. doi: 10.1152/ajpheart.00012.2007. Epub 2007 Aug 24.
5
The role of mitochondria in protection of the heart by preconditioning.线粒体在预处理对心脏的保护作用中的角色。
Biochim Biophys Acta. 2007 Aug;1767(8):1007-31. doi: 10.1016/j.bbabio.2007.05.008. Epub 2007 Jun 2.
6
Oxygen-dependent regulation of mitochondrial respiration by hypoxia-inducible factor 1.缺氧诱导因子1对线粒体呼吸的氧依赖性调节
Biochem J. 2007 Jul 1;405(1):1-9. doi: 10.1042/BJ20070389.
7
Reduced force generating capacity in myocytes from chronically ischemic, hibernating myocardium.
Circ Res. 2007 Feb 2;100(2):229-37. doi: 10.1161/01.RES.0000257829.07721.57. Epub 2007 Jan 18.
8
Mitochondrial adaptations within chronically ischemic swine myocardium.慢性缺血猪心肌内的线粒体适应性变化
J Mol Cell Cardiol. 2006 Dec;41(6):980-8. doi: 10.1016/j.yjmcc.2006.07.008. Epub 2006 Aug 22.
9
HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption.缺氧诱导因子-1通过积极下调线粒体氧消耗来介导对缺氧的适应。
Cell Metab. 2006 Mar;3(3):187-97. doi: 10.1016/j.cmet.2006.01.012.
10
Different mechanisms of mitochondrial proton leak in ischaemia/reperfusion injury and preconditioning: implications for pathology and cardioprotection.缺血/再灌注损伤和预处理中线粒体质子泄漏的不同机制：对病理学和心脏保护的影响。
Biochem J. 2006 May 1;395(3):611-8. doi: 10.1042/BJ20051927.